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Nature Structural & Molecular Biology|December 8, 2009
A promiscuous alpha-helical motif anchors viral hijackers and substrate receptors to the CUL4-DDB1 ubiquitin ligase machineryTi Li, Eva I Robert, Pieter C van Breugel, et al.Journal of Virology|June 1, 2018
Smc5/6 Antagonism by HBx Is an Evolutionarily Conserved Function of Hepatitis B Virus Infection in MammalsFabien Abdul, Fabien Filleton, Laetitia Gerossier, et al.Science Advances|September 12, 2020
Histone exchange is associated with activator function at transcribed promoters and with repression at histone lociSari Kassem, Paolo Ferrari, Amanda L Hughes, et al.Journal of Hepatology|March 8, 2011
Hepatitis B virus X protein is essential to initiate and maintain virus replication after infectionJulie Lucifora, Silke Arzberger, David Durantel, et al.Hepatology (Baltimore, Md.)|June 30, 2012
Hepatitis B virus X protein stimulates gene expression selectively from extrachromosomal DNA templatesPieter C van Breugel, Eva I Robert, Henrik Mueller, et al.Nature Structural & Molecular Biology|September 13, 2022
Smc5/6 silences episomal transcription by a three-step functionFabien Abdul, Aurélie Diman, Bastien Baechler, et al.Hepatology (Baltimore, Md.)|September 11, 2008
Hepatitis B virus X protein affects S phase progression leading to chromosome segregation defects by binding to damaged DNA binding protein 1Silvia Martin-Lluesma, Céline Schaeffer, Eva Isabelle Robert, et al.Nature|March 18, 2016
Hepatitis B virus X protein identifies the Smc5/6 complex as a host restriction factorAdrien Decorsière, Henrik Mueller, Pieter C van Breugel, et al.Plos One|January 18, 2017
The Smc5/6 Complex Restricts HBV when Localized to ND10 without Inducing an Innate Immune Response and Is Counteracted by the HBV X Protein Shortly after InfectionCongrong Niu, Christine M Livingston, Li Li, et al.Pageof 2