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Journal of Neurology|February 8, 2017
Functional impairment in patients with myotonic dystrophy type 1 can be assessed by an ataxia rating scale (SARA)Giovanni DiPaolo, Cecilia Jimenez-Moreno, Nikoletta Nikolenko, et al.Neuropediatrics|September 27, 2015
Testosterone Treatment of Pubertal Delay in Duchenne Muscular DystrophyClaire L Wood, Tim D Cheetham, Michela Guglieri, et al.MDM Policy & Practice|June 15, 2026
Evaluating the Impact of Different Natural History Modeling Methods on Cost-Effectiveness Decisions: A Case Study in Duchenne Muscular DystrophyJonathan Broomfield, Keith R Abrams, Michael J Crowther, et al.Neurology|July 4, 2014
The burden of Duchenne muscular dystrophy: an international, cross-sectional studyErik Landfeldt, Peter Lindgren, Christopher F Bell, et al.Journal of Neuromuscular Diseases|February 13, 2016
Compliance to Care Guidelines for Duchenne Muscular DystrophyErik Landfeldt, Peter Lindgren, Christopher F Bell, et al.Rheumatology Advances in Practice|August 14, 2024
The development of pGALSplus: evaluating feasibility and acceptability of an assessment to facilitate the identification and triage of children with musculoskeletal presentationsVicky Mercer, Nicola Smith, Michela Guglieri, et al.Brain : a Journal of Neurology|October 8, 2004
Wild-type bone marrow cells ameliorate the phenotype of SOD1-G93A ALS mice and contribute to CNS, heart and skeletal muscle tissuesStefania Corti, Federica Locatelli, Chiara Donadoni, et al.Plos One|September 4, 2019
Categorising trajectories and individual item changes of the North Star Ambulatory Assessment in patients with Duchenne muscular dystrophyFrancesco Muntoni, Joana Domingos, Adnan Y Manzur, et al.The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|September 24, 2004
Improvement of combined FISH and immunofluorescence to trace the fate of somatic stem cells after transplantationChiara Donadoni, Stefania Corti, Federica Locatelli, et al.Human Gene Therapy Methods|October 19, 2012
Exon skipping quantification by quantitative reverse-transcription polymerase chain reaction in Duchenne muscular dystrophy patients treated with the antisense oligomer eteplirsenKaren Anthony, Lucy Feng, Virginia Arechavala-Gomeza, et al.Pageof 14