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Biochemical Society Transactions|May 17, 2008
Can donor splice site recognition occur without the involvement of U1 snRNP?Michela Raponi, Diana BaralleNucleic Acids Research|December 19, 2006
Reduced splicing efficiency induced by synonymous substitutions may generate a substrate for natural selection of new splicing isoforms: the case of CFTR exon 12Michela Raponi, Francisco E Baralle, Franco PaganiProceedings of the National Academy of Sciences of the United States of America|April 21, 2005
Synonymous mutations in CFTR exon 12 affect splicing and are not neutral in evolutionFranco Pagani, Michela Raponi, Francisco E BaralleInternational Journal of Molecular Sciences|July 25, 2014
BRCA1 EXON 11, a CERES (composite regulatory element of splicing) element involved in splice regulationClaudia Tammaro, Michela Raponi, David I Wilson, et al.Biochemical Society Transactions|July 24, 2012
BRCA1 exon 11 alternative splicing, multiple functions and the association with cancerClaudia Tammaro, Michela Raponi, David I Wilson, et al.The FEBS Journal|March 19, 2009
Low U1 snRNP dependence at the NF1 exon 29 donor splice siteMichela Raponi, Emanuele Buratti, Elisa Dassie, et al.RNA Biology|March 25, 2014
BRCA1 exon 11 a model of long exon splicing regulationMichela Raponi, Lindsay D Smith, Marco Silipo, et al.Plos One|May 23, 2012
Evolutionary constraint helps unmask a splicing regulatory region in BRCA1 exon 11Michela Raponi, Andrew G L Douglas, Claudia Tammaro, et al.The FEBS Journal|November 20, 2008
Polypyrimidine tract binding protein regulates alternative splicing of an aberrant pseudoexon in NF1Michela Raponi, Emanuele Buratti, Miriam Llorian, et al.Human Mutation|February 11, 2011
Prediction of single-nucleotide substitutions that result in exon skipping: identification of a splicing silencer in BRCA1 exon 6Michela Raponi, Jana Kralovicova, Ellen Copson, et al.Pageof 2