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Michele H Jacob

Showing results (11-20 of 23) with videos related to

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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 16, 2005
A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde mannerSiamak Rassadi, Arjun Krishnaswamy, Brigitte Pié, et al.
Channels (Austin, Tex.)|January 8, 2014
Alternative splice isoforms of small conductance calcium-activated SK2 channels differ in molecular interactions and surface levelsElizabeth Storer Scholl, Antonella Pirone, Daniel H Cox, et al.
Journal of Neurochemistry|May 25, 2010
Alexa Fluor 546-ArIB[V11L;V16A] is a potent ligand for selectively labeling alpha 7 nicotinic acetylcholine receptorsArik J Hone, Paul Whiteaker, Jesse L Mohn, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 20, 2010
The postsynaptic adenomatous polyposis coli (APC) multiprotein complex is required for localizing neuroligin and neurexin to neuronal nicotinic synapses in vivoMadelaine M Rosenberg, Fang Yang, Jesse L Mohn, et al.
Molecular and Cellular Neurosciences|April 15, 2008
Adenomatous polyposis coli plays a key role, in vivo, in coordinating assembly of the neuronal nicotinic postsynaptic complexMadelaine M Rosenberg, Fang Yang, Monica Giovanni, et al.
EMBO Molecular Medicine|August 5, 2024
Inhibition of GSK3α,β rescues cognitive phenotypes in a preclinical mouse model of CTNNB1 syndromeJonathan M Alexander, Leeanne Vazquez-Ramirez, Crystal Lin, et al.
Human Molecular Genetics|May 28, 2019
Learning impairments and molecular changes in the brain caused by β-catenin lossRobert J Wickham, Jonathan M Alexander, Lillian W Eden, et al.
Neurobiology of Disease|November 17, 2016
APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizuresAntonella Pirone, Jonathan Alexander, Lauren A Lau, et al.
Molecular Therapy. Nucleic Acids|February 19, 2025
Use of polyadenosine tail mimetics to enhance mRNA expression from genes associated with haploinsufficiency disordersBahareh Torkzaban, Yining Zhu, Christian Lopez, et al.
Frontiers in Molecular Neuroscience|April 12, 2021
Deletion of BDNF in Pax2 Lineage-Derived Interneuron Precursors in the Hindbrain Hampers the Proportion of Excitation/Inhibition, Learning, and BehaviorPhilipp Eckert, Philine Marchetta, Marie K Manthey, et al.
Pageof 3

Showing results (11-20 of 23) with videos related to

Sort By:
Pageof 3
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 16, 2005
A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde mannerSiamak Rassadi, Arjun Krishnaswamy, Brigitte Pié, et al.
Channels (Austin, Tex.)|January 8, 2014
Alternative splice isoforms of small conductance calcium-activated SK2 channels differ in molecular interactions and surface levelsElizabeth Storer Scholl, Antonella Pirone, Daniel H Cox, et al.
Journal of Neurochemistry|May 25, 2010
Alexa Fluor 546-ArIB[V11L;V16A] is a potent ligand for selectively labeling alpha 7 nicotinic acetylcholine receptorsArik J Hone, Paul Whiteaker, Jesse L Mohn, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 20, 2010
The postsynaptic adenomatous polyposis coli (APC) multiprotein complex is required for localizing neuroligin and neurexin to neuronal nicotinic synapses in vivoMadelaine M Rosenberg, Fang Yang, Jesse L Mohn, et al.
Molecular and Cellular Neurosciences|April 15, 2008
Adenomatous polyposis coli plays a key role, in vivo, in coordinating assembly of the neuronal nicotinic postsynaptic complexMadelaine M Rosenberg, Fang Yang, Monica Giovanni, et al.
EMBO Molecular Medicine|August 5, 2024
Inhibition of GSK3α,β rescues cognitive phenotypes in a preclinical mouse model of CTNNB1 syndromeJonathan M Alexander, Leeanne Vazquez-Ramirez, Crystal Lin, et al.
Human Molecular Genetics|May 28, 2019
Learning impairments and molecular changes in the brain caused by β-catenin lossRobert J Wickham, Jonathan M Alexander, Lillian W Eden, et al.
Neurobiology of Disease|November 17, 2016
APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizuresAntonella Pirone, Jonathan Alexander, Lauren A Lau, et al.
Molecular Therapy. Nucleic Acids|February 19, 2025
Use of polyadenosine tail mimetics to enhance mRNA expression from genes associated with haploinsufficiency disordersBahareh Torkzaban, Yining Zhu, Christian Lopez, et al.
Frontiers in Molecular Neuroscience|April 12, 2021
Deletion of BDNF in Pax2 Lineage-Derived Interneuron Precursors in the Hindbrain Hampers the Proportion of Excitation/Inhibition, Learning, and BehaviorPhilipp Eckert, Philine Marchetta, Marie K Manthey, et al.
Pageof 3