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Scientific Reports|November 10, 2017
Delivery of Native Proteins into C. elegans Using a Transduction Protocol Based on Lipid VesiclesMichele Perni, Francesco A Aprile, Sam Casford, et al.Scientific Reports|July 14, 2021
Exogenous misfolded protein oligomers can cross the intestinal barrier and cause a disease phenotype in C. elegansMichele Perni, Benedetta Mannini, Catherine K Xu, et al.ACS Chemical Neuroscience|July 11, 2018
Stabilization and Characterization of Cytotoxic Aβ<sub>40</sub> Oligomers Isolated from an Aggregation Reaction in the Presence of Zinc IonsBenedetta Mannini, Johnny Habchi, Sean Chia, et al.Communications Biology|July 8, 2021
Two human metabolites rescue a C. elegans model of Alzheimer's disease via a cytosolic unfolded protein responsePriyanka Joshi, Michele Perni, Ryan Limbocker, et al.Frontiers in Molecular Neuroscience|April 21, 2017
Chaperones as Suppressors of Protein Misfolded Oligomer ToxicityBenedetta Mannini, Fabrizio ChitiNpj Aging|December 20, 2024
Anti-aging properties of the aminosterols of the dogfish sharkDenise Barbut, Michele Perni, Michael ZasloffACS Chemical Neuroscience|July 9, 2024
Preparation and Characterization of Zn(II)-Stabilized Aβ<sub>42</sub> OligomersAlicia González Díaz, Rodrigo Cataldi, Benedetta Mannini, et al.Scientific Reports|September 18, 2020
A rationally designed bicyclic peptide remodels Aβ42 aggregation in vitro and reduces its toxicity in a worm model of Alzheimer's diseaseTatsuya Ikenoue, Francesco A Aprile, Pietro Sormanni, et al.Proceedings of the National Academy of Sciences of the United States of America|December 25, 2016
Systematic development of small molecules to inhibit specific microscopic steps of Aβ42 aggregation in Alzheimer's diseaseJohnny Habchi, Sean Chia, Ryan Limbocker, et al.Pageof 7