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Mikhail A Kolpakov

Showing results (1-10 of 16) with videos related to

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Circulation Research|March 22, 2008
Sympathetic activation causes focal adhesion signaling alteration in early compensated volume overload attributable to isolated mitral regurgitation in the dogAbdelkarim Sabri, Khadija Rafiq, Rachid Seqqat, et al.
The Journal of Biological Chemistry|May 13, 2006
Role of protein-tyrosine phosphatase SHP2 in focal adhesion kinase down-regulation during neutrophil cathepsin G-induced cardiomyocytes anoikisKhadija Rafiq, Mikhail A Kolpakov, Malika Abdelfettah, et al.
The Journal of Biological Chemistry|December 29, 2011
c-Cbl ubiquitin ligase regulates focal adhesion protein turnover and myofibril degeneration induced by neutrophil protease cathepsin GKhadija Rafiq, Jianfen Guo, Liudmila Vlasenko, et al.
Basic Research in Cardiology|September 16, 2017
Dual inhibition of cathepsin G and chymase reduces myocyte death and improves cardiac remodeling after myocardial ischemia reperfusion injuryBahman Hooshdaran, Mikhail A Kolpakov, Xinji Guo, et al.
Journal of Molecular and Cellular Cardiology|June 29, 2019
Intracardiac administration of neutrophil protease cathepsin G activates noncanonical inflammasome pathway and promotes inflammation and pathological remodeling in non-injured heartSonni-Ali Miller, Mikhail A Kolpakov, Xinji Guo, et al.
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|December 13, 2019
Inflammatory Serine Proteases Play a Critical Role in the Early Pathogenesis of Diabetic CardiomyopathyMikhail A Kolpakov, Kunal Sikder, Amrita Sarkar, et al.
Journal of Molecular and Cellular Cardiology|May 22, 2012
Beta1-adrenergic receptors promote focal adhesion signaling downregulation and myocyte apoptosis in acute volume overloadRachid Seqqat, Xinji Guo, Khadija Rafiq, et al.
Molecular Neurobiology|July 6, 2018
Hyperglycemia-Driven Neuroinflammation Compromises BBB Leading to Memory Loss in Both Diabetes Mellitus (DM) Type 1 and Type 2 Mouse ModelsSlava Rom, Viviana Zuluaga-Ramirez, Sachin Gajghate, et al.
Journal of Molecular and Cellular Cardiology|September 1, 2009
Pleiotropic effects of neutrophils on myocyte apoptosis and left ventricular remodeling during early volume overloadMikhail A Kolpakov, Rachid Seqqat, Khadija Rafiq, et al.
Journal of Molecular and Cellular Cardiology|December 9, 2015
Protease-activated receptor 4 deficiency offers cardioprotection after acute ischemia reperfusion injuryMikhail A Kolpakov, Khadija Rafiq, Xinji Guo, et al.
Pageof 2

Showing results (1-10 of 16) with videos related to

Sort By:
Pageof 2
Circulation Research|March 22, 2008
Sympathetic activation causes focal adhesion signaling alteration in early compensated volume overload attributable to isolated mitral regurgitation in the dogAbdelkarim Sabri, Khadija Rafiq, Rachid Seqqat, et al.
The Journal of Biological Chemistry|May 13, 2006
Role of protein-tyrosine phosphatase SHP2 in focal adhesion kinase down-regulation during neutrophil cathepsin G-induced cardiomyocytes anoikisKhadija Rafiq, Mikhail A Kolpakov, Malika Abdelfettah, et al.
The Journal of Biological Chemistry|December 29, 2011
c-Cbl ubiquitin ligase regulates focal adhesion protein turnover and myofibril degeneration induced by neutrophil protease cathepsin GKhadija Rafiq, Jianfen Guo, Liudmila Vlasenko, et al.
Basic Research in Cardiology|September 16, 2017
Dual inhibition of cathepsin G and chymase reduces myocyte death and improves cardiac remodeling after myocardial ischemia reperfusion injuryBahman Hooshdaran, Mikhail A Kolpakov, Xinji Guo, et al.
Journal of Molecular and Cellular Cardiology|June 29, 2019
Intracardiac administration of neutrophil protease cathepsin G activates noncanonical inflammasome pathway and promotes inflammation and pathological remodeling in non-injured heartSonni-Ali Miller, Mikhail A Kolpakov, Xinji Guo, et al.
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|December 13, 2019
Inflammatory Serine Proteases Play a Critical Role in the Early Pathogenesis of Diabetic CardiomyopathyMikhail A Kolpakov, Kunal Sikder, Amrita Sarkar, et al.
Journal of Molecular and Cellular Cardiology|May 22, 2012
Beta1-adrenergic receptors promote focal adhesion signaling downregulation and myocyte apoptosis in acute volume overloadRachid Seqqat, Xinji Guo, Khadija Rafiq, et al.
Molecular Neurobiology|July 6, 2018
Hyperglycemia-Driven Neuroinflammation Compromises BBB Leading to Memory Loss in Both Diabetes Mellitus (DM) Type 1 and Type 2 Mouse ModelsSlava Rom, Viviana Zuluaga-Ramirez, Sachin Gajghate, et al.
Journal of Molecular and Cellular Cardiology|September 1, 2009
Pleiotropic effects of neutrophils on myocyte apoptosis and left ventricular remodeling during early volume overloadMikhail A Kolpakov, Rachid Seqqat, Khadija Rafiq, et al.
Journal of Molecular and Cellular Cardiology|December 9, 2015
Protease-activated receptor 4 deficiency offers cardioprotection after acute ischemia reperfusion injuryMikhail A Kolpakov, Khadija Rafiq, Xinji Guo, et al.
Pageof 2