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Journal of Biochemistry|July 9, 2016
The double knockout of Bach1 and Bach2 in mice reveals shared compensatory mechanisms in regulating alveolar macrophage function and lung surfactant homeostasisRisa Ebina-Shibuya, Miki Watanabe-Matsui, Mitsuyo Matsumoto, et al.
Journal of Biochemistry|May 22, 2016
Charge-state-distribution analysis of Bach2 intrinsically disordered heme binding regionTomoji Suenaga, Miki Watanabe-Matsui, Tamami Uejima, et al.
Nucleic Acids Research|February 21, 2015
Restriction-modification system with methyl-inhibited base excision and abasic-site cleavage activitiesMasaki Fukuyo, Toshiaki Nakano, Yingbiao Zhang, et al.
Blood|March 30, 2011
Heme regulates B-cell differentiation, antibody class switch, and heme oxygenase-1 expression in B cells as a ligand of Bach2Miki Watanabe-Matsui, Akihiko Muto, Toshitaka Matsui, et al.
Biochemical and Biophysical Research Communications|May 13, 2021
Polymorphisms in glia maturation factor β gene are markers of cellulose ether effectiveness in prion-infected miceKenta Teruya, Ayumi Oguma, Keita Arai, et al.
The Biochemical Journal|February 21, 2018
Phosphorylation of BACH1 switches its function from transcription factor to mitotic chromosome regulator and promotes its interaction with HMMRJie Li, Hiroki Shima, Hironari Nishizawa, et al.
The Journal of Biological Chemistry|December 2, 2015
The Transcription Factor Bach2 Is Phosphorylated at Multiple Sites in Murine B Cells but a Single Site Prevents Its Nuclear LocalizationRyo Ando, Hiroki Shima, Toru Tamahara, et al.
Nature Immunology|October 27, 2014
The transcription repressors Bach2 and Bach1 promote B cell development by repressing the myeloid programAri Itoh-Nakadai, Reina Hikota, Akihiko Muto, et al.
International Journal of Molecular Sciences|April 27, 2024
TANK Binding Kinase 1 Promotes BACH1 Degradation through Both Phosphorylation-Dependent and -Independent Mechanisms without Relying on Heme and FBXO22Liang Liu, Mitsuyo Matsumoto, Miki Watanabe-Matsui, et al.
Iscience|January 6, 2025
Heme regulates protein interactions and phosphorylation of BACH2 intrinsically disordered region in humoral responseMiki Watanabe-Matsui, Shun Kadoya, Kei Segawa, et al.
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