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Adipocyte|May 24, 2013
The GSK-3β/Fyn/Nrf2 pathway in fibroblasts and wounds of type 2 diabetes: On the road to an evidence-based therapy of non-healing woundsMilad S BitarInternational Journal of Molecular Sciences|February 6, 2019
Diabetes Impairs Angiogenesis and Induces Endothelial Cell Senescence by Up-Regulating Thrombospondin-CD47-Dependent SignalingMilad S BitarDisease Models & Mechanisms|February 25, 2012
ROS constitute a convergence nexus in the development of IGF1 resistance and impaired wound healing in a rat model of type 2 diabetesMilad S Bitar, Fahd Al-MullaAmerican Journal of Physiology. Endocrinology and Metabolism|September 1, 2011
A defect in Nrf2 signaling constitutes a mechanism for cellular stress hypersensitivity in a genetic rat model of type 2 diabetesMilad S Bitar, Fahd Al-MullaJournal of Biomedical Science|August 19, 2004
Antioxidants attenuate diabetes-induced activation of peroxisomal functions in the rat kidneyGursev S Dhaunsi, Milad S BitarDisease Models & Mechanisms|November 9, 2014
Upregulation of CREM/ICER suppresses wound endothelial CRE-HIF-1α-VEGF-dependent signaling and impairs angiogenesis in type 2 diabetesMilad S Bitar, Fahd Al-MullaInternational Journal of Molecular Sciences|September 28, 2023
Activation of 5-HT1A Receptors Normalizes the Overexpression of Presynaptic 5-HT1A Receptors and Alleviates Diabetic Neuropathic PainNeha Munawar, Milad S Bitar, Willias MasochaPain|April 16, 1998
Attenuation of IGF-1 antinociceptive action and a reduction in spinal cord gene expression of its receptor in experimental diabetesMilad S Bitar, T C W PilcherBiotechniques|November 25, 2004
Comparison of two probe preparation methods using long oligonucleotide microarraysFahd Al-Mulla, Raba' Al-Tamimi, Milad S BitarLife Sciences|June 7, 2005
Oxidative stress--mediated alterations in glucose dynamics in a genetic animal model of type II diabetesMilad S Bitar, Eyad Al-Saleh, Fahd Al-MullaPageof 4