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Milena Pinto

Showing results (21-30 of 33) with videos related to

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EMBO Molecular Medicine|August 21, 2024
Restoration of defective oxidative phosphorylation to a subset of neurons prevents mitochondrial encephalopathyBrittni R Walker, Lise-Michelle Theard, Milena Pinto, et al.
Human Molecular Genetics|September 24, 2021
Enhanced glycolysis and GSK3 inactivation promote brain metabolic adaptations following neuronal mitochondrial stressSofia Garcia, Amy Saldana-Caboverde, Mir Anwar, et al.
Human Molecular Genetics|June 14, 2013
Transient systemic mtDNA damage leads to muscle wasting by reducing the satellite cell poolXiao Wang, Alicia M Pickrell, Susana G Rossi, et al.
Molecular Therapy. Nucleic Acids|February 26, 2024
mitoTALEN reduces the mutant mtDNA load in neuronsSandra R Bacman, Jose Domingo Barrera-Paez, Milena Pinto, et al.
The Journal of Biological Chemistry|April 2, 2011
Tumor necrosis factor receptor-associated factor 6 (TRAF6) associates with huntingtin protein and promotes its atypical ubiquitination to enhance aggregate formationSilvia Zucchelli, Federica Marcuzzi, Marta Codrich, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 14, 2026
Dopaminergic neurons preferentially accumulate mtDNA rearrangementsTania Arguello, Christian D Alcalde Pretel, Nadee Nissanka, et al.
Nature Medicine|October 7, 2018
Author Correction: MitoTALEN reduces mutant mtDNA load and restores tRNA<sup>Ala</sup> levels in a mouse model of heteroplasmic mtDNA mutationSandra R Bacman, Johanna H K Kauppila, Claudia V Pereira, et al.
Nature Medicine|September 26, 2018
MitoTALEN reduces mutant mtDNA load and restores tRNA<sup>Ala</sup> levels in a mouse model of heteroplasmic mtDNA mutationSandra R Bacman, Johanna H K Kauppila, Claudia V Pereira, et al.
Cell Death and Differentiation|December 3, 2016
Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent mannerMilena Pinto, Alicia M Pickrell, Xiao Wang, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 22, 2017
Mitochondrial DNA Double-Strand Breaks in Oligodendrocytes Cause Demyelination, Axonal Injury, and CNS InflammationPernille M Madsen, Milena Pinto, Shreyans Patel, et al.
Pageof 4

Showing results (21-30 of 33) with videos related to

Sort By:
Pageof 4
EMBO Molecular Medicine|August 21, 2024
Restoration of defective oxidative phosphorylation to a subset of neurons prevents mitochondrial encephalopathyBrittni R Walker, Lise-Michelle Theard, Milena Pinto, et al.
Human Molecular Genetics|September 24, 2021
Enhanced glycolysis and GSK3 inactivation promote brain metabolic adaptations following neuronal mitochondrial stressSofia Garcia, Amy Saldana-Caboverde, Mir Anwar, et al.
Human Molecular Genetics|June 14, 2013
Transient systemic mtDNA damage leads to muscle wasting by reducing the satellite cell poolXiao Wang, Alicia M Pickrell, Susana G Rossi, et al.
Molecular Therapy. Nucleic Acids|February 26, 2024
mitoTALEN reduces the mutant mtDNA load in neuronsSandra R Bacman, Jose Domingo Barrera-Paez, Milena Pinto, et al.
The Journal of Biological Chemistry|April 2, 2011
Tumor necrosis factor receptor-associated factor 6 (TRAF6) associates with huntingtin protein and promotes its atypical ubiquitination to enhance aggregate formationSilvia Zucchelli, Federica Marcuzzi, Marta Codrich, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 14, 2026
Dopaminergic neurons preferentially accumulate mtDNA rearrangementsTania Arguello, Christian D Alcalde Pretel, Nadee Nissanka, et al.
Nature Medicine|October 7, 2018
Author Correction: MitoTALEN reduces mutant mtDNA load and restores tRNA<sup>Ala</sup> levels in a mouse model of heteroplasmic mtDNA mutationSandra R Bacman, Johanna H K Kauppila, Claudia V Pereira, et al.
Nature Medicine|September 26, 2018
MitoTALEN reduces mutant mtDNA load and restores tRNA<sup>Ala</sup> levels in a mouse model of heteroplasmic mtDNA mutationSandra R Bacman, Johanna H K Kauppila, Claudia V Pereira, et al.
Cell Death and Differentiation|December 3, 2016
Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent mannerMilena Pinto, Alicia M Pickrell, Xiao Wang, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 22, 2017
Mitochondrial DNA Double-Strand Breaks in Oligodendrocytes Cause Demyelination, Axonal Injury, and CNS InflammationPernille M Madsen, Milena Pinto, Shreyans Patel, et al.
Pageof 4