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Journal of the American Heart Association
|
November 7, 2012
Tafazzin knockdown in mice leads to a developmental cardiomyopathy with early diastolic dysfunction preceding myocardial noncompaction
Colin K L Phoon, Devrim Acehan, Michael Schlame, et al.
The Journal of Cell Biology
|
March 28, 2019
Extramitochondrial cardiolipin suggests a novel function of mitochondria in spermatogenesis
Mindong Ren, Yang Xu, Hediye Erdjument-Bromage, et al.
Nature Chemical Biology
|
June 28, 2016
Loss of protein association causes cardiolipin degradation in Barth syndrome
Yang Xu, Colin K L Phoon, Bob Berno, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
November 20, 2003
Rab27b is associated with fusiform vesicles and may be involved in targeting uroplakins to urothelial apical membranes
Yanru Chen, Xuemei Guo, Fang-Ming Deng, et al.
The Journal of Biological Chemistry
|
August 25, 2009
Characterization of tafazzin splice variants from humans and fruit flies
Yang Xu, Shali Zhang, Ashim Malhotra, et al.
Mitochondrion
|
December 31, 2008
Distinct effects of tafazzin deletion in differentiated and undifferentiated mitochondria
Devrim Acehan, Zaza Khuchua, Riekelt H Houtkooper, et al.
Biorxiv : the Preprint Server for Biology
|
February 14, 2024
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction and leads to increased glucose uptake in tafazzin-deficient cells
Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, et al.
Scientific Reports
|
May 20, 2024
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction in tafazzin-deficient cells
Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, et al.
Orphanet Journal of Rare Diseases
|
March 11, 2017
The PPAR pan-agonist bezafibrate ameliorates cardiomyopathy in a mouse model of Barth syndrome
Yan Huang, Corey Powers, Victoria Moore, et al.
Page
of 5
Search research articles
Search
Showing results (41-50 of 49) with videos related to
Sort By:
Page
of 5
You have reached the last page of results.
This site can display upto 49 results.
Journal of the American Heart Association
|
November 7, 2012
Tafazzin knockdown in mice leads to a developmental cardiomyopathy with early diastolic dysfunction preceding myocardial noncompaction
Colin K L Phoon, Devrim Acehan, Michael Schlame, et al.
The Journal of Cell Biology
|
March 28, 2019
Extramitochondrial cardiolipin suggests a novel function of mitochondria in spermatogenesis
Mindong Ren, Yang Xu, Hediye Erdjument-Bromage, et al.
Nature Chemical Biology
|
June 28, 2016
Loss of protein association causes cardiolipin degradation in Barth syndrome
Yang Xu, Colin K L Phoon, Bob Berno, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
November 20, 2003
Rab27b is associated with fusiform vesicles and may be involved in targeting uroplakins to urothelial apical membranes
Yanru Chen, Xuemei Guo, Fang-Ming Deng, et al.
The Journal of Biological Chemistry
|
August 25, 2009
Characterization of tafazzin splice variants from humans and fruit flies
Yang Xu, Shali Zhang, Ashim Malhotra, et al.
Mitochondrion
|
December 31, 2008
Distinct effects of tafazzin deletion in differentiated and undifferentiated mitochondria
Devrim Acehan, Zaza Khuchua, Riekelt H Houtkooper, et al.
Biorxiv : the Preprint Server for Biology
|
February 14, 2024
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction and leads to increased glucose uptake in tafazzin-deficient cells
Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, et al.
Scientific Reports
|
May 20, 2024
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction in tafazzin-deficient cells
Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, et al.
Orphanet Journal of Rare Diseases
|
March 11, 2017
The PPAR pan-agonist bezafibrate ameliorates cardiomyopathy in a mouse model of Barth syndrome
Yan Huang, Corey Powers, Victoria Moore, et al.
Page
of 5