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Virology|May 5, 2009
Deletion of the AcMNPV core gene ac109 results in budded virions that are non-infectiousMinggang Fang, Yingchao Nie, David A TheilmannJournal of Virology|July 13, 2007
Autographa californica multiple nucleopolyhedrovirus EXON0 (ORF141) is required for efficient egress of nucleocapsids from the nucleusMinggang Fang, Xiaojiang Dai, David A TheilmannVirology|October 23, 2010
Autographa californica multiple nucleopolyhedrovirus core gene ac92 (p33) is required for efficient budded virus productionYingchao Nie, Minggang Fang, David A TheilmannVirology|January 20, 2009
AcMNPV AC16 (DA26, BV/ODV-E26) regulates the levels of IE0 and IE1 and binds to both proteins via a domain located within the acidic transcriptional activation domainYingchao Nie, Minggang Fang, David A TheilmannVirology|January 22, 2009
AcMNPV EXON0 (AC141) which is required for the efficient egress of budded virus nucleocapsids interacts with beta-tubulinMinggang Fang, Yingchao Nie, David A TheilmannMolecular Cell|September 16, 2014
The BRAF oncoprotein functions through the transcriptional repressor MAFG to mediate the CpG Island Methylator phenotypeMinggang Fang, Jianhong Ou, Lloyd Hutchinson, et al.Virology|March 4, 2008
Identification of AcMNPV EXON0 (ac141) domains required for efficient production of budded virus, dimerization and association with BV/ODV-C42 and FP25Minggang Fang, Yingchao Nie, Xiaojiang Dai, et al.Proceedings of the National Academy of Sciences of the United States of America|January 21, 2016
Common BRAF(V600E)-directed pathway mediates widespread epigenetic silencing in colorectal cancer and melanomaMinggang Fang, Lloyd Hutchinson, April Deng, et al.Frontiers in Immunology|September 16, 2024
Unlocking the potential of iPSC-derived immune cells: engineering iNK and iT cells for cutting-edge immunotherapyMinggang Fang, Alexander Allen, Chong Luo, et al.Journal of Virology|January 27, 2012
Analysis of the autographa californica multiple nucleopolyhedrovirus overlapping gene pair lef3 and ac68 reveals that AC68 is a per os infectivity factor and that LEF3 is critical, but not essential, for virus replicationYingchao Nie, Minggang Fang, Martin A Erlandson, et al.Pageof 3