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Circulation Research|August 25, 2017
Activation of γ2-AMPK Suppresses Ribosome Biogenesis and Protects Against Myocardial Ischemia/Reperfusion InjuryYang Cao, Naveen Bojjireddy, Maengjo Kim, et al.Circulation Research|March 12, 2013
Broad suppression of NADPH oxidase activity exacerbates ischemia/reperfusion injury through inadvertent downregulation of hypoxia-inducible factor-1α and upregulation of peroxisome proliferator-activated receptor-αShouji Matsushima, Junya Kuroda, Tetsuro Ago, et al.Plos One|July 30, 2011
Comparative analysis of mRNA isoform expression in cardiac hypertrophy and development reveals multiple post-transcriptional regulatory modulesJi Yeon Park, Wencheng Li, Dinghai Zheng, et al.American Journal of Physiology. Heart and Circulatory Physiology|July 27, 2019
Both gain and loss of Nampt function promote pressure overload-induced heart failureJaemin Byun, Shin-Ichi Oka, Nobushige Imai, et al.Antioxidants & Redox Signaling|June 4, 2014
The role of SUMO-1 in cardiac oxidative stress and hypertrophyAhyoung Lee, Dongtak Jeong, Shinichi Mitsuyama, et al.American Journal of Physiology. Cell Physiology|July 24, 2009
Cytochrome c oxidase III as a mechanism for apoptosis in heart failure following myocardial infarctionChanggong Wu, Lin Yan, Christophe Depre, et al.The EMBO Journal|April 28, 2017
A postprandial FGF19-SHP-LSD1 regulatory axis mediates epigenetic repression of hepatic autophagySangwon Byun, Young-Chae Kim, Yang Zhang, et al.Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|December 15, 2015
Detrimental role of pericyte Nox4 in the acute phase of brain ischemiaAtaru Nishimura, Tetsuro Ago, Junya Kuroda, et al.Journal of Molecular and Cellular Cardiology|June 6, 2026
YAP mediates activation of the serine synthesis pathway in the heart during pressure overloadChengchen Hu, Peiyong Zhai, Xiaoyang Su, et al.The Journal of Clinical Investigation|December 29, 2020
YAP plays a crucial role in the development of cardiomyopathy in lysosomal storage diseasesShohei Ikeda, Jihoon Nah, Akihiro Shirakabe, et al.Pageof 34