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International Journal of Endocrinology|January 19, 2016
Paraoxonase 2 Induces a Phenotypic Switch in Macrophage Polarization Favoring an M2 Anti-Inflammatory StateMarie Koren-Gluzer, Mira Rosenblat, Tony HayekArteriosclerosis, Thrombosis, and Vascular Biology|November 1, 2003
Decreased macrophage paraoxonase 2 expression in patients with hypercholesterolemia is the result of their increased cellular cholesterol content: effect of atorvastatin therapyMira Rosenblat, Tony Hayek, Khetam Hussein, et al.Lipids|June 27, 2016
Nitro-Oleic Acid Reduces J774A.1 Macrophage Oxidative Status and Triglyceride Mass: Involvement of Paraoxonase2 and Triglyceride Metabolizing EnzymesMira Rosenblat, Oren Rom, Nina Volkova, et al.Atherosclerosis|February 22, 2005
Paraoxonase 1 (PON1) enhances HDL-mediated macrophage cholesterol efflux via the ABCA1 transporter in association with increased HDL binding to the cells: a possible role for lysophosphatidylcholineMira Rosenblat, Jacob Vaya, Diana Shih, et al.Clinical Chemistry and Laboratory Medicine|March 28, 2002
Serum paraoxonase activity and the extent of lipid peroxidation are not affected by increased levels of human apolipoprotein A-I: studies in transgenic miceMira Rosenblat, Orit Grunfeld, Tony Hayek, et al.Atherosclerosis|July 30, 2011
Paraoxonase 1 (PON1) inhibits monocyte-to-macrophage differentiationMira Rosenblat, Nina Volkova, John Ward, et al.Journal of Cardiovascular Pharmacology|June 20, 2002
Valsartan therapy has additive anti-oxidative effect to that of fluvastatin therapy against low-density lipoprotein oxidation: studies in hypercholesterolemic and hypertensive patientsOsamah Hussein, Julia Shneider, Mira Rosenblat, et al.Biofactors (Oxford, England)|October 11, 2012
Triglyceride accumulation in macrophages upregulates paraoxonase 2 (PON2) expression via ROS-mediated JNK/c-Jun signaling pathway activationMira Rosenblat, Nina Volkova, Nicole Paland, et al.Journal of Agricultural and Food Chemistry|March 2, 2006
Pomegranate byproduct administration to apolipoprotein e-deficient mice attenuates atherosclerosis development as a result of decreased macrophage oxidative stress and reduced cellular uptake of oxidized low-density lipoproteinMira Rosenblat, Nina Volkova, Raymond Coleman, et al.Atherosclerosis|June 26, 2007
Anti-oxidant and anti-atherogenic properties of liposomal glutathione: studies in vitro, and in the atherosclerotic apolipoprotein E-deficient miceMira Rosenblat, Nina Volkova, Raymond Coleman, et al.Pageof 6