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Alzheimer'S Research & Therapy|November 1, 2013
Autophagic/lysosomal dysfunction in Alzheimer's diseaseMiranda E Orr, Salvatore OddoNeurobiology of Aging|August 20, 2013
Accumulation of C-terminal fragments of transactive response DNA-binding protein 43 leads to synaptic loss and cognitive deficits in human TDP-43 transgenic miceDavid X Medina, Miranda E Orr, Salvatore OddoNeurobiology of Aging|January 14, 2014
Mammalian target of rapamycin hyperactivity mediates the detrimental effects of a high sucrose diet on Alzheimer's disease pathologyMiranda E Orr, Angelica Salinas, Rochelle Buffenstein, et al.Frontiers in Bioscience (Scholar Edition)|December 29, 2011
The role of mTOR signaling in Alzheimer diseaseSalvatore OddoJournal of Cellular and Molecular Medicine|February 13, 2008
The ubiquitin-proteasome system in Alzheimer's diseaseSalvatore OddoAlzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 23, 2025
Basic Science and PathogenesisMiranda E OrrJournal of Alzheimer'S Disease : JAD|March 10, 2024
A Need for Refined Senescence Biomarkers and Measures of Senolytics in the BrainMiranda E OrrTrends in Molecular Medicine|April 13, 2005
Alzheimer's disease: Abeta, tau and synaptic dysfunctionFrank M LaFerla, Salvatore OddoJournal of Physiology, Paris|February 2, 2006
The role of nicotinic acetylcholine receptors in Alzheimer's diseaseSalvatore Oddo, Frank M LaFerlaThe American Journal of Pathology|November 10, 2011
Cognitive decline typical of frontotemporal lobar degeneration in transgenic mice expressing the 25-kDa C-terminal fragment of TDP-43Antonella Caccamo, Smita Majumder, Salvatore OddoPageof 16