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Journal of Medical Virology|February 28, 2023
MAML1-induced HPV E6 oncoprotein stability is required for cellular proliferation and migration of cervical tumor-derived cellsJosipa Skelin, Anamaria Đukić, Vedrana Filić, et al.The FEBS Journal|March 4, 2021
Words of Advice: How to be a good Principal InvestigatorLawrence BanksScientific Reports|January 14, 2021
Inhibition of kinase IKKβ suppresses cellular abnormalities induced by the human papillomavirus oncoprotein HPV 18E6Mojgan Padash Barmchi, Miranda Thomas, Jayashree V Thatte, et al.Apoptosis : an International Journal on Programmed Cell Death|December 28, 2006
Cleavage of MAGI-1, a tight junction PDZ protein, by caspases is an important step for cell-cell detachment in apoptosisUros Gregorc, Saska Ivanova, Miranda Thomas, et al.Pathogens (Basel, Switzerland)|February 23, 2020
HPV Oncoproteins and the Ubiquitin Proteasome System: A Signature of Malignancy?Anamaria Đukić, Lucija Lulić, Miranda Thomas, et al.The Journal of General Virology|January 25, 2019
Oncogenicitiy Comparison of Human Papillomavirus Type 52 E6 VariantsTsz On Lai, Siaw Shi Boon, Priscilla Ty Law, et al.Journal of Virology|October 11, 2019
PDZ Domain-Containing Protein NHERF-2 Is a Novel Target of Human Papillomavirus 16 (HPV-16) and HPV-18Nathaniel Edward Bennett Saidu, Vedrana Filić, Miranda Thomas, et al.Journal of Virology|December 3, 2010
A systematic analysis of human papillomavirus (HPV) E6 PDZ substrates identifies MAGI-1 as a major target of HPV type 16 (HPV-16) and HPV-18 whose loss accompanies disruption of tight junctionsChristian Kranjec, Lawrence BanksMutation Research. Reviews in Mutation Research|May 23, 2017
Molecular mechanisms underlying human papillomavirus E6 and E7 oncoprotein-induced cell transformationSuruchi Mittal, Lawrence BanksPageof 18