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Journal of Virology|August 25, 2017
Human Papillomavirus 16 (HPV-16), HPV-18, and HPV-31 E6 Override the Normal Phosphoregulation of E6AP Enzymatic ActivityJayashree Thatte, Lawrence BanksJournal of Virology|November 2, 2012
SNX17 facilitates infection with diverse papillomavirus typesMartina Bergant, Lawrence BanksInternational Journal of Cancer|November 26, 2003
p53 polymorphic variants at codon 72 exert different effects on cell cycle progressionDavid Pim, Lawrence BanksThe Journal of Biological Chemistry|August 7, 2003
Regulation of the discs large tumor suppressor by a phosphorylation-dependent interaction with the beta-TrCP ubiquitin ligase receptorFiamma Mantovani, Lawrence BanksViruses|October 26, 2021
The Not-So-Good, the Bad and the Ugly: HPV E5, E6 and E7 Oncoproteins in the Orchestration of CarcinogenesisOm Basukala, Lawrence BanksAPMIS : Acta Pathologica, Microbiologica, Et Immunologica Scandinavica|June 18, 2010
Interaction of viral oncoproteins with cellular target molecules: infection with high-risk vs low-risk human papillomavirusesDavid Pim, Lawrence BanksMethods in Molecular Medicine|December 15, 2005
Transformation assays for HPV oncoproteinsPaola Massimi, Lawrence BanksMbio|October 18, 2022
HPV-16 E7 Interacts with the Endocytic Machinery via the AP2 Adaptor μ2 SubunitOm Basukala, Oscar Trejo-Cerro, Michael P Myers, et al.The Journal of General Virology|February 28, 2019
Acquisition of a phospho-acceptor site enhances HPV E6 PDZ-binding motif functional promiscuityVanessa Sarabia-Vega, Lawrence BanksAntiviral Therapy|November 13, 2004
HPV proteins as targets for therapeutic interventionHelena Sterlinko Grm, Lawrence BanksPageof 18