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Methods in Molecular Biology (Clifton, N.J.)|May 12, 2021
Studying Metabolic Abnormalities in the Costello Syndrome HRAS G12V Mouse Model: Isolation of Mouse Embryonic Fibroblasts and Their In Vitro Adipocyte DifferentiationMiray Fidan, Saravanakkumar Chennappan, Ion Cristian CirsteaMechanisms of Ageing and Development|December 14, 2020
Senescence in RASopathies, a possible novel contributor to a complex pathophenoypeMelanie Engler, Miray Fidan, Sayantan Nandi, et al.Frontiers in Immunology|July 30, 2019
Cdk5 Deletion Enhances the Anti-inflammatory Potential of GC-Mediated GR Activation During InflammationPauline Pfänder, Miray Fidan, Ute Burret, et al.Scientific Reports|February 25, 2025
Impaired MC3T3-E1 osteoblast differentiation triggered by oncogenic HRAS is rescued by the farnesyltransferase inhibitor TipifarnibYannik Andrasch, Moses Munene Ireri, Jonas Gander, et al.Frontiers in Cell and Developmental Biology|November 4, 2022
Increased osteoclastogenesis contributes to bone loss in the Costello syndrome Hras G12V mouse modelSayantan Nandi, Saravanakkumar Chennappan, Yannik Andrasch, et al.Wound Repair and Regeneration : Official Publication of the Wound Healing Society [And] the European Tissue Repair Society|October 2, 2019
Local and transient inhibition of p21 expression ameliorates age-related delayed wound healingDongsheng Jiang, Juliane C de Vries, Jana Muschhammer, et al.Human Molecular Genetics|November 28, 2018
Dominant Noonan syndrome-causing LZTR1 mutations specifically affect the Kelch domain substrate-recognition surface and enhance RAS-MAPK signalingMarialetizia Motta, Miray Fidan, Emanuele Bellacchio, et al.Science Signaling|March 22, 2022
The glucocorticoid receptor associates with RAS complexes to inhibit cell proliferation and tumor growthBozhena Caratti, Miray Fidan, Giorgio Caratti, et al.Pageof 1