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Elife|January 22, 2020
HOPS recognizes each SNARE, assembling ternary trans-complexes for rapid fusion upon engagement with the 4th SNAREHongki Song, Amy S Orr, Miriam Lee, et al.Progress in Lipid Research|April 24, 2013
The role of the gut in reverse cholesterol transport--focus on the enterocyteMiriam Lee-Rueckert, Francisco Blanco-Vaca, Petri T Kovanen, et al.Journal of Lipid Research|February 4, 2003
Apolipoprotein composition and particle size affect HDL degradation by chymase: effect on cellular cholesterol effluxMiriam Lee, Petri T Kovanen, Gabriella Tedeschi, et al.Atherosclerosis|August 15, 2009
Mast cell-dependent proteolytic modification of HDL particles during anaphylactic shock in the mouse reduces their ability to induce cholesterol efflux from macrophage foam cells ex vivoIlona Judström, Heli Jukkola, Jari Metso, et al.Journal of Lipid Research|May 12, 2006
Absence of endogenous phospholipid transfer protein impairs ABCA1-dependent efflux of cholesterol from macrophage foam cellsMiriam Lee-Rueckert, Riikka Vikstedt, Jari Metso, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|August 13, 2010
Acidic extracellular environments strongly impair ABCA1-mediated cholesterol efflux from human macrophage foam cellsMiriam Lee-Rueckert, Jani Lappalainen, Hannele Leinonen, et al.Scientific Reports|March 2, 2021
Cholesterol loading suppresses the atheroinflammatory gene polarization of human macrophages induced by colony stimulating factorsJani Lappalainen, Nicolas Yeung, Su D Nguyen, et al.The Journal of Biological Chemistry|January 1, 2004
Depletion of pre-beta-high density lipoprotein by human chymase impairs ATP-binding cassette transporter A1- but not scavenger receptor class B type I-mediated lipid efflux to high density lipoproteinElda Favari, Miriam Lee, Laura Calabresi, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|December 17, 2002
Mast cell tryptase degrades HDL and blocks its function as an acceptor of cellular cholesterolMiriam Lee, Christian P Sommerhoff, Arnold von Eckardstein, et al.Atherosclerosis|September 16, 2011
High-density lipoproteins (HDL) are present in stenotic aortic valves and may interfere with the mechanisms of valvular calcificationJaakko I Lommi, Petri T Kovanen, Matti Jauhiainen, et al.Pageof 7