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Current Opinion in Lipidology|September 5, 2015
The mast cell as a pluripotent HDL-modifying effector in atherogenesis: from in vitro to in vivo significanceMiriam Lee-Rueckert, Petri T Kovanen
Current Opinion in Lipidology|September 2, 2011
Extracellular modifications of HDL in vivo and the emerging concept of proteolytic inactivation of preβ-HDLMiriam Lee-Rueckert, Petri T Kovanen
Biochimica Et Biophysica Acta|March 13, 2016
HDL functionality in reverse cholesterol transport--Challenges in translating data emerging from mouse models to human diseaseMiriam Lee-Rueckert, Joan Carles Escola-Gil, Petri T Kovanen
Current Medicinal Chemistry|August 24, 2019
Molecular Insights into the Mechanisms Underlying the Cholesterol- Lowering Effects of PhytosterolsLídia Cedó, Marta Farràs, Miriam Lee-Rueckert, et al.
Frontiers in Cardiovascular Medicine|March 3, 2022
Lipid-Laden Macrophages and Inflammation in Atherosclerosis and Cancer: An Integrative ViewMiriam Lee-Rueckert, Jani Lappalainen, Petri T Kovanen, et al.
Journal of Lipid Research|November 10, 2007
Association of cholesteryl ester transfer protein with HDL particles reduces its proteolytic inactivation by mast cell chymaseMiriam Lee-Rueckert, Riikka Vikstedt, Jari Metso, et al.
Atherosclerosis|April 3, 2016
Conversion of human M-CSF macrophages into foam cells reduces their proinflammatory responses to classical M1-polarizing activationRafaela F da Silva, Jani Lappalainen, Miriam Lee-Rueckert, et al.
Seminars in Cancer Biology|June 22, 2025
Lipids and lipoproteins in the interstitial tissue fluid regulate the formation of dysfunctional tissue-resident macrophages: Implications for atherogenic, tumorigenic, and obesogenic processesMiriam Lee-Rueckert, Matti Jauhiainen, Petri T Kovanen, et al.
Progress in Lipid Research|April 24, 2013
The role of the gut in reverse cholesterol transport--focus on the enterocyteMiriam Lee-Rueckert, Francisco Blanco-Vaca, Petri T Kovanen, et al.
Atherosclerosis|August 15, 2009
Mast cell-dependent proteolytic modification of HDL particles during anaphylactic shock in the mouse reduces their ability to induce cholesterol efflux from macrophage foam cells ex vivoIlona Judström, Heli Jukkola, Jari Metso, et al.
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