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Mirko Baruscotti

Showing results (31-40 of 58) with videos related to

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The Journal of Physiology|April 19, 2003
Heteromeric HCN1-HCN4 channels: a comparison with native pacemaker channels from the rabbit sinoatrial nodeClaudia Altomare, Benedetta Terragni, Chiara Brioschi, et al.
Phytomedicine : International Journal of Phytotherapy and Phytopharmacology|February 5, 2020
High content screening identifies licoisoflavone A as a bioactive compound of Tongmaiyangxin Pills to restrain cardiomyocyte hypertrophy via activating Sirt3Rui Guo, Ningning Liu, Hao Liu, et al.
Vascular Pharmacology|July 10, 2024
NOD-1 activation increases the spontaneous activity and the I(f) current of murine sinoatrial node cellsSerena Canzolino, Giorgia Bertoli, Samira Bakhouba, et al.
Veterinary Journal (London, England : 1997)|September 9, 2010
Multinucleated giant cells with an osteoclast phenotype derived from caprine peripheral blood mononuclear cellsGiuliano Pisoni, Patrizia D'Amelio, Francesca Sassi, et al.
Vascular Pharmacology|July 10, 2024
A gain of function mutation in PITX2c gene linked to atrial fibrillation alters mitochondrial function and membrane excitabilityPatrizia Benzoni, Lorenzo Da Dalt, Alessandro Cospito, et al.
European Heart Journal|February 10, 2017
A gain-of-function mutation in the cardiac pacemaker HCN4 channel increasing cAMP sensitivity is associated with familial Inappropriate Sinus TachycardiaMirko Baruscotti, Annalisa Bucchi, Raffaella Milanesi, et al.
Scientific Reports|April 16, 2016
INaP selective inhibition reverts precocious inter- and motorneurons hyperexcitability in the Sod1-G93R zebrafish ALS modelLorena Benedetti, Anna Ghilardi, Elsa Rottoli, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 2, 2011
Recessive loss-of-function mutation in the pacemaker HCN2 channel causing increased neuronal excitability in a patient with idiopathic generalized epilepsyJacopo C DiFrancesco, Andrea Barbuti, Raffaella Milanesi, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 12, 2011
Deep bradycardia and heart block caused by inducible cardiac-specific knockout of the pacemaker channel gene Hcn4Mirko Baruscotti, Annalisa Bucchi, Carlo Viscomi, et al.
Vascular Pharmacology|July 10, 2024
In-vitro studies of the NaV1.5 S805L Brugada mutation: The resting cell voltage is a critical element in determining the pathological or physiological phenotype of the currentDavid Molla, Anthony Frosio, Giorgia Bertoli, et al.
Pageof 6

Showing results (31-40 of 58) with videos related to

Sort By:
Pageof 6
The Journal of Physiology|April 19, 2003
Heteromeric HCN1-HCN4 channels: a comparison with native pacemaker channels from the rabbit sinoatrial nodeClaudia Altomare, Benedetta Terragni, Chiara Brioschi, et al.
Phytomedicine : International Journal of Phytotherapy and Phytopharmacology|February 5, 2020
High content screening identifies licoisoflavone A as a bioactive compound of Tongmaiyangxin Pills to restrain cardiomyocyte hypertrophy via activating Sirt3Rui Guo, Ningning Liu, Hao Liu, et al.
Vascular Pharmacology|July 10, 2024
NOD-1 activation increases the spontaneous activity and the I(f) current of murine sinoatrial node cellsSerena Canzolino, Giorgia Bertoli, Samira Bakhouba, et al.
Veterinary Journal (London, England : 1997)|September 9, 2010
Multinucleated giant cells with an osteoclast phenotype derived from caprine peripheral blood mononuclear cellsGiuliano Pisoni, Patrizia D'Amelio, Francesca Sassi, et al.
Vascular Pharmacology|July 10, 2024
A gain of function mutation in PITX2c gene linked to atrial fibrillation alters mitochondrial function and membrane excitabilityPatrizia Benzoni, Lorenzo Da Dalt, Alessandro Cospito, et al.
European Heart Journal|February 10, 2017
A gain-of-function mutation in the cardiac pacemaker HCN4 channel increasing cAMP sensitivity is associated with familial Inappropriate Sinus TachycardiaMirko Baruscotti, Annalisa Bucchi, Raffaella Milanesi, et al.
Scientific Reports|April 16, 2016
INaP selective inhibition reverts precocious inter- and motorneurons hyperexcitability in the Sod1-G93R zebrafish ALS modelLorena Benedetti, Anna Ghilardi, Elsa Rottoli, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 2, 2011
Recessive loss-of-function mutation in the pacemaker HCN2 channel causing increased neuronal excitability in a patient with idiopathic generalized epilepsyJacopo C DiFrancesco, Andrea Barbuti, Raffaella Milanesi, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 12, 2011
Deep bradycardia and heart block caused by inducible cardiac-specific knockout of the pacemaker channel gene Hcn4Mirko Baruscotti, Annalisa Bucchi, Carlo Viscomi, et al.
Vascular Pharmacology|July 10, 2024
In-vitro studies of the NaV1.5 S805L Brugada mutation: The resting cell voltage is a critical element in determining the pathological or physiological phenotype of the currentDavid Molla, Anthony Frosio, Giorgia Bertoli, et al.
Pageof 6