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Science Advances|June 8, 2022
An isogenic panel of <i>App</i> knock-in mouse models: Profiling β-secretase inhibition and endosomal abnormalitiesNaoto Watamura, Kaori Sato, Gen Shiihashi, et al.Eneuro|November 26, 2024
Tau Accumulation Induces Microglial State Alterations in Alzheimer's Disease Model MiceKenichi Nagata, Shoko Hashimoto, Daisuke Joho, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 29, 2025
The Role of Neprilysin and Insulin-Degrading Enzyme in the Etiology of Sporadic Alzheimer's DiseaseTakahiro Morito, Shoko Hashimoto, Risa Takamura, et al.Journal of Alzheimer'S Disease : JAD|November 19, 2025
Somatostatin receptor subtypes 1 and 4 regulate neprilysin, the major amyloid-β degrading enzyme in brainPer Nilsson, Karin Sörgjerd, Naomasa Kakiya, et al.Life Science Alliance|September 30, 2024
Metabolic resistance of Aβ3pE-42, a target epitope of the anti-Alzheimer therapeutic antibody, donanemabNobuhisa Iwata, Satoshi Tsubuki, Misaki Sekiguchi, et al.Acta Neuropathologica Communications|July 5, 2022
Increased CSF-decorin predicts brain pathological changes driven by Alzheimer's Aβ amyloidosisRicheng Jiang, Una Smailovic, Hazal Haytural, et al.Pageof 2