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Journal of Human Genetics
|
May 27, 2016
De novo MEIS2 mutation causes syndromic developmental delay with persistent gastro-esophageal reflux
Atsushi Fujita, Bertrand Isidor, Hugues Piloquet, et al.
Brain & Development
|
June 7, 2021
Novel ALG12 variants and hydronephrosis in siblings with impaired N-glycosylation
Takuya Hiraide, Yoshinao Wada, Tomoko Matsubayashi, et al.
Nature Genetics
|
August 17, 2010
A genome-wide association study identifies four susceptibility loci for keloid in the Japanese population
Mitsuko Nakashima, Suyoun Chung, Atsushi Takahashi, et al.
Plos One
|
April 15, 2020
CCNB2 and AURKA overexpression may cause atypical mitosis in Japanese cortisol-producing adrenocortical carcinoma with TP53 somatic variant
Akira Ikeya, Mitsuko Nakashima, Miho Yamashita, et al.
Neurogenetics
|
June 8, 2014
Novel compound heterozygous PIGT mutations caused multiple congenital anomalies-hypotonia-seizures syndrome 3
Mitsuko Nakashima, Hirofumi Kashii, Yoshiko Murakami, et al.
Journal of Human Genetics
|
January 30, 2015
SPTAN1 encephalopathy: distinct phenotypes and genotypes
Jun Tohyama, Mitsuko Nakashima, Shin Nabatame, et al.
Journal of Neurology, Neurosurgery, and Psychiatry
|
May 22, 2015
Homozygous p.V116* mutation in C12orf65 results in Leigh syndrome
Eri Imagawa, Aviva Fattal-Valevski, Ori Eyal, et al.
Epileptic Disorders : International Epilepsy Journal with Videotape
|
August 8, 2020
A de novo GABRB2 variant associated with myoclonic status epilepticus and rhythmic high-amplitude delta with superimposed (poly) spikes (RHADS)
Aiko Nishikawa, Yui Otani, Susumu Ito, et al.
Journal of Human Genetics
|
June 26, 2015
Novel compound heterozygous LIAS mutations cause glycine encephalopathy
Yoshinori Tsurusaki, Ryuta Tanaka, Shino Shimada, et al.
Brain & Development
|
October 21, 2015
High prevalence of genetic alterations in early-onset epileptic encephalopathies associated with infantile movement disorders
Yu Kobayashi, Jun Tohyama, Mitsuhiro Kato, et al.
Page
of 21
Search research articles
Search
Showing results (71-80 of 208) with videos related to
Sort By:
Page
of 21
Journal of Human Genetics
|
May 27, 2016
De novo MEIS2 mutation causes syndromic developmental delay with persistent gastro-esophageal reflux
Atsushi Fujita, Bertrand Isidor, Hugues Piloquet, et al.
Brain & Development
|
June 7, 2021
Novel ALG12 variants and hydronephrosis in siblings with impaired N-glycosylation
Takuya Hiraide, Yoshinao Wada, Tomoko Matsubayashi, et al.
Nature Genetics
|
August 17, 2010
A genome-wide association study identifies four susceptibility loci for keloid in the Japanese population
Mitsuko Nakashima, Suyoun Chung, Atsushi Takahashi, et al.
Plos One
|
April 15, 2020
CCNB2 and AURKA overexpression may cause atypical mitosis in Japanese cortisol-producing adrenocortical carcinoma with TP53 somatic variant
Akira Ikeya, Mitsuko Nakashima, Miho Yamashita, et al.
Neurogenetics
|
June 8, 2014
Novel compound heterozygous PIGT mutations caused multiple congenital anomalies-hypotonia-seizures syndrome 3
Mitsuko Nakashima, Hirofumi Kashii, Yoshiko Murakami, et al.
Journal of Human Genetics
|
January 30, 2015
SPTAN1 encephalopathy: distinct phenotypes and genotypes
Jun Tohyama, Mitsuko Nakashima, Shin Nabatame, et al.
Journal of Neurology, Neurosurgery, and Psychiatry
|
May 22, 2015
Homozygous p.V116* mutation in C12orf65 results in Leigh syndrome
Eri Imagawa, Aviva Fattal-Valevski, Ori Eyal, et al.
Epileptic Disorders : International Epilepsy Journal with Videotape
|
August 8, 2020
A de novo GABRB2 variant associated with myoclonic status epilepticus and rhythmic high-amplitude delta with superimposed (poly) spikes (RHADS)
Aiko Nishikawa, Yui Otani, Susumu Ito, et al.
Journal of Human Genetics
|
June 26, 2015
Novel compound heterozygous LIAS mutations cause glycine encephalopathy
Yoshinori Tsurusaki, Ryuta Tanaka, Shino Shimada, et al.
Brain & Development
|
October 21, 2015
High prevalence of genetic alterations in early-onset epileptic encephalopathies associated with infantile movement disorders
Yu Kobayashi, Jun Tohyama, Mitsuhiro Kato, et al.
Page
of 21