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Biomedicines|June 26, 2025
Role of Cav1.3 Channels in Brain-Heart Interactions: An Unexpected JourneyJean-Baptiste Reisqs, Yvonne Sleiman, Michael Cupelli, et al.
Frontiers in Cardiovascular Medicine|August 21, 2023
Ethnic and racial differences in Asian populations with ion channelopathies associated with sudden cardiac deathSahil Zaveri, Yongxia Sarah Qu, Mohamed Chahine, et al.
Frontiers in Bioscience (Scholar Edition)|December 29, 2011
Regulation of cardiac excitability by protein kinase C isozymesJulio Cesar Batista Ferreira, Daria Mochly-Rosen, Mohamed Boutjdir
JACC. Basic to Translational Science|July 10, 2023
Fir(e)ing the Rhythm: Inflammatory Cytokines and Cardiac ArrhythmiasPietro Enea Lazzerini, Antonio Abbate, Mohamed Boutjdir, et al.
Biochemical and Biophysical Research Communications|February 9, 2007
Expression of skeletal muscle Na(V)1.4 Na channel isoform in canine cardiac Purkinje myocytesYongxia Qu, Eddy Karnabi, Mohamed Chahine, et al.
Frontiers in Physiology|February 15, 2021
Voltage/Calcium Uncoupling Underlies Sustained Torsade de Pointes Ventricular Tachyarrhythmia in an Experimental Model of Long QT SyndromeHerman D Himel, Michael Cupelli, Mohamed Boutjdir, et al.
Molecular Diagnosis & Therapy|April 17, 2026
Therapeutic Strategies Targeting the Molecular Pathogenesis of Myotonic Dystrophy Type 1: Current Status and Future DirectionsMohamed Chahine, Vamsi Krishna Murthy Ginjupalli, Dominic Jauvin, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 16, 2006
Protein kinase C activation inhibits Cav1.3 calcium channel at NH2-terminal serine 81 phosphorylation siteGhayath Baroudi, Yongxia Qu, Omar Ramadan, et al.
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