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Biomedicines|June 26, 2025
Role of Cav1.3 Channels in Brain-Heart Interactions: An Unexpected JourneyJean-Baptiste Reisqs, Yvonne Sleiman, Michael Cupelli, et al.Frontiers in Cardiovascular Medicine|August 21, 2023
Ethnic and racial differences in Asian populations with ion channelopathies associated with sudden cardiac deathSahil Zaveri, Yongxia Sarah Qu, Mohamed Chahine, et al.Frontiers in Bioscience (Scholar Edition)|December 29, 2011
Regulation of cardiac excitability by protein kinase C isozymesJulio Cesar Batista Ferreira, Daria Mochly-Rosen, Mohamed BoutjdirJACC. Basic to Translational Science|July 10, 2023
Fir(e)ing the Rhythm: Inflammatory Cytokines and Cardiac ArrhythmiasPietro Enea Lazzerini, Antonio Abbate, Mohamed Boutjdir, et al.Biochemical and Biophysical Research Communications|February 9, 2007
Expression of skeletal muscle Na(V)1.4 Na channel isoform in canine cardiac Purkinje myocytesYongxia Qu, Eddy Karnabi, Mohamed Chahine, et al.Frontiers in Physiology|February 15, 2021
Voltage/Calcium Uncoupling Underlies Sustained Torsade de Pointes Ventricular Tachyarrhythmia in an Experimental Model of Long QT SyndromeHerman D Himel, Michael Cupelli, Mohamed Boutjdir, et al.Molecular Diagnosis & Therapy|April 17, 2026
Therapeutic Strategies Targeting the Molecular Pathogenesis of Myotonic Dystrophy Type 1: Current Status and Future DirectionsMohamed Chahine, Vamsi Krishna Murthy Ginjupalli, Dominic Jauvin, et al.American Journal of Physiology. Heart and Circulatory Physiology|September 16, 2006
Protein kinase C activation inhibits Cav1.3 calcium channel at NH2-terminal serine 81 phosphorylation siteGhayath Baroudi, Yongxia Qu, Omar Ramadan, et al.Stem Cell Research|February 5, 2023
Generation of four myotonic dystrophy type 1 patient iPSC lines (CBRCULi002-A, CBRCULi003-A, CBRCULi004-A, CBRCULi005-A) and a control (CBRCULi001-A) derived from lymphoblastoids cell linesDominic Jauvin, Marion Pierre, Mohamed Boutjdir, et al.Stem Cell Research|March 15, 2024
Generation of three myotonic dystrophy type 1 patient iPSC lines (CBRCULi018-A, CBRCULi019-A, CBRCULi020-A) derived from lymphoblastoid cell lines for disease modelling and therapeutic researchMarion Pierre, Dominic Jauvin, Jack Puymirat, et al.Pageof 12