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Brain Research. Molecular Brain Research|October 26, 2002
Sodium channel Na(v)1.6 is expressed along nonmyelinated axons and it contributes to conductionJoel A Black, Muthukrishnan Renganathan, Stephen G WaxmanNeurobiology of Pain (Cambridge, Mass.)|April 22, 2026
Biophysical dissection of nociceptor hyperexcitability caused by a Nav1.8 gain-of-function mutation linked to severe painDmytro V Vasylyev, Peng Zhao, Stephen G WaxmanMolecular Pain|August 19, 2006
Sodium channel expression in the ventral posterolateral nucleus of the thalamus after peripheral nerve injuryPeng Zhao, Stephen G Waxman, Bryan C HainsGlia|December 31, 2008
Sodium channel activity modulates multiple functions in microgliaJoel A Black, Shujun Liu, Stephen G WaxmanMultiple Sclerosis (Houndmills, Basingstoke, England)|September 7, 2012
Nav1.5 sodium channels in macrophages in multiple sclerosis lesionsJoel A Black, Jia Newcombe, Stephen G WaxmanThe Journal of Physiology|October 17, 2023
Ih current stabilizes excitability in rodent DRG neurons and reverses hyperexcitability in a nociceptive neuron model of inherited neuropathic painDmytro V Vasylyev, Shujun Liu, Stephen G WaxmanGlia|February 27, 2016
Sodium channels in astroglia and microgliaLaura W Pappalardo, Joel A Black, Stephen G WaxmanMolecular Neurobiology|November 14, 2002
Structure of the sodium channel gene SCN11A: evidence for intron-to-exon conversion model and implications for gene evolutionSulayman D Dib-Hajj, Lynda Tyrrell, Stephen G WaxmanExperimental Brain Research|August 5, 2003
GTP gamma S increases Nav1.8 current in small-diameter dorsal root ganglia neuronsCarl Y Saab, Theodore R Cummins, Stephen G WaxmanThe Journal of Comparative Neurology|June 10, 2003
Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injuryBryan C Hains, Joel A Black, Stephen G WaxmanPageof 42