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Neurology|September 22, 2006
Inherited erythermalgia: limb pain from an S4 charge-neutral Na channelopathyJin-Sung Choi, Sulayman D Dib-Hajj, Stephen G WaxmanPain Medicine (Malden, Mass.)|October 13, 2009
Voltage-gated sodium channels: therapeutic targets for painSulayman D Dib-Hajj, Joel A Black, Stephen G WaxmanJournal of Neurophysiology|November 17, 2006
Differential slow inactivation and use-dependent inhibition of Nav1.8 channels contribute to distinct firing properties in IB4+ and IB4- DRG neuronsJin-Sung Choi, Sulayman D Dib-Hajj, Stephen G WaxmanNature Reviews. Neuroscience|August 6, 2015
NaV1.9: a sodium channel linked to human painSulayman D Dib-Hajj, Joel A Black, Stephen G WaxmanTrends in Pharmacological Sciences|July 11, 2025
Ion channels as therapeutic targets in osteoarthritisRenpeng Zhou, Wei Hu, Stephen G Waxman, et al.Experimental Neurology|May 24, 2011
Nav1.7 accumulates and co-localizes with phosphorylated ERK1/2 within transected axons in early experimental neuromasAnna-Karin Persson, Andreas Gasser, Joel A Black, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 12, 2003
Calmodulin binds to the C terminus of sodium channels Nav1.4 and Nav1.6 and differentially modulates their functional propertiesRaimund I Herzog, Chuanju Liu, Stephen G Waxman, et al.The Journal of General Physiology|October 8, 2024
Interplay of Nav1.8 and Nav1.7 channels drives neuronal hyperexcitability in neuropathic painDmytro V Vasylyev, Peng Zhao, Betsy R Schulman, et al.The Journal of Biological Chemistry|September 30, 2006
Size matters: Erythromelalgia mutation S241T in Nav1.7 alters channel gatingAngelika Lampert, Sulayman D Dib-Hajj, Lynda Tyrrell, et al.Journal of Neuropathology and Experimental Neurology|September 7, 2007
Sodium channel expression within chronic multiple sclerosis plaquesJoel A Black, Jia Newcombe, Bruce D Trapp, et al.Pageof 42