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Nature Reviews. Rheumatology|August 9, 2024
Ion channels in osteoarthritis: emerging roles and potential targetsRenpeng Zhou, Wenyu Fu, Dmytro Vasylyev, et al.Journal of Neurophysiology|April 7, 2006
Calmodulin regulates current density and frequency-dependent inhibition of sodium channel Nav1.8 in DRG neuronsJin-Sung Choi, Andy Hudmon, Stephen G Waxman, et al.Neurobiology of Pain (Cambridge, Mass.)|May 14, 2019
Brain activity associated with pain in inherited erythromelalgia: stimulus-free pain engages brain areas involved in valuation and learningPaul Geha, Betsy R Schulman, Sulayman D Dib-Hajj, et al.Neuroscience Letters|November 9, 2020
Mini-review - Sodium channels and beyond in peripheral nerve disease: Modulation by cytokines and their effector protein kinasesXiaoyang Cheng, Jin-Sung Choi, Stephen G Waxman, et al.FEBS Letters|August 12, 2004
Functional role of the C-terminus of voltage-gated sodium channel Na(v)1.8Jin-Sung Choi, Lynda Tyrrell, Stephen G Waxman, et al.Novartis Foundation Symposium|January 5, 2002
Diverse functions and dynamic expression of neuronal sodium channelsStephen G Waxman, Theodore R Cummins, Joel A Black, et al.Nature Reviews. Neuroscience|December 13, 2012
The Na(V)1.7 sodium channel: from molecule to manSulayman D Dib-Hajj, Yang Yang, Joel A Black, et al.Molecular Neurobiology|April 17, 2008
Locomotor dysfunction and pain: the scylla and charybdis of fiber sprouting after spinal cord injuryRonald Deumens, Elbert A J Joosten, Stephen G Waxman, et al.Glia|April 18, 2014
Voltage-gated sodium channel Nav 1.5 contributes to astrogliosis in an in vitro model of glial injury via reverse Na+ /Ca2+ exchangeLaura W Pappalardo, Omar A Samad, Joel A Black, et al.Molecular Pain|November 9, 2012
Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal hornJoel A Black, Noémie Frézel, Sulayman D Dib-Hajj, et al.Pageof 42