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Biophysical Journal|September 18, 2007
Negative charges at protein kinase C sites of troponin I stabilize the inactive state of actinMohit C Mathur, Tomoyoshi Kobayashi, Joseph M ChalovichBiophysical Journal|March 18, 2009
Some cardiomyopathy-causing troponin I mutations stabilize a functional intermediate actin stateMohit C Mathur, Tomoyoshi Kobayashi, Joseph M ChalovichBiochemistry|July 26, 2016
The Cardiomyopathy Mutation, R146G Troponin I, Stabilizes the Intermediate "C" State of Regulated Actin under High- and Low-Free Ca(2+) ConditionsDylan Johnson, Mohit C Mathur, Tomoyoshi Kobayashi, et al.Biochemical and Biophysical Research Communications|February 8, 2011
Several cardiomyopathy causing mutations on tropomyosin either destabilize the active state of actomyosin or alter the binding properties of tropomyosinMohit C Mathur, P Bryant Chase, Joseph M ChalovichBiophysical Journal|June 21, 2012
The C-terminus of troponin T is essential for maintaining the inactive state of regulated actinAndrew J Franklin, Tamatha Baxley, Tomoyoshi Kobayashi, et al.Journal of Muscle Research and Cell Motility|June 9, 2012
Disease causing mutations of troponin alter regulated actin state distributionsJoseph M ChalovichJournal of Muscle Research and Cell Motility|April 28, 2012
Michael Bárány: a recollectionJoseph M ChalovichMethods in Molecular Biology (Clifton, N.J.)|October 24, 2007
Equilibrium binding of proteins to F-actinJoseph M ChalovichBiochemistry|October 27, 2004
Ca2+-calmodulin regulates fesselin-induced actin polymerizationMechthild Schroeter, Joseph M ChalovichJournal of Muscle Research and Cell Motility|February 2, 2006
Smooth muscle alpha-actinin binds tightly to fesselin and attenuates its activity toward actin polymerizationMinh Pham, Joseph M ChalovichPageof 8