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Biorxiv : the Preprint Server for Biology|September 30, 2024
Fibroblasts Regulate the Transformation Potential of Human Papillomavirus-positive KeratinocytesClaire D James, Rachel L Lewis, Austin J Witt, et al.
Tumour Virus Research|December 12, 2024
Fibroblasts regulate the transcriptional signature of human papillomavirus-positive keratinocytesClaire D James, Rachel L Lewis, Austin J Witt, et al.
Virology|September 4, 2022
Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage responseChristian T Fontan, Apurva T Prabhakar, Xu Wang, et al.
Msphere|December 10, 2020
Human Papillomavirus 16 (HPV16) E2 Repression of TWIST1 Transcription Is a Potential Mediator of HPV16 Cancer OutcomesChristian T Fontan, Dipon Das, Molly L Bristol, et al.
Journal of Virology|July 26, 2022
Interaction with TopBP1 Is Required for Human Papillomavirus 16 E2 Plasmid Segregation/Retention Function during MitosisApurva T Prabhakar, Claire D James, Dipon Das, et al.
Msphere|January 10, 2020
Human Papillomavirus 16 E6 and E7 Synergistically Repress Innate Immune Gene TranscriptionClaire D James, Christian T Fontan, Raymonde Otoa, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
The Utility of Fibroblast Co-culture for the Maintenance of Episomes in Human Papillomavirus-Associated Cancer ModelsAustin J Witt, Rachel L Lewis, Xu Wang, et al.
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