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Elife|October 19, 2013
A component of the mir-17-92 polycistronic oncomir promotes oncogene-dependent apoptosisVirginie Olive, Erich Sabio, Margaux J Bennett, et al.Molecular Cancer Therapeutics|November 6, 2023
Genetic Silencing of AKT Induces Melanoma Cell Death via mTOR SuppressionGennie L Parkman, Tursun Turapov, David A Kircher, et al.BMC Medicine|June 6, 2017
Comprehensive DNA methylation study identifies novel progression-related and prognostic markers for cutaneous melanomaJasper Wouters, Miguel Vizoso, Anna Martinez-Cardus, et al.Cancer Research|December 5, 2024
Targeting the PREX2/RAC1/PI3Kβ Signaling Axis Confers Sensitivity to Clinically Relevant Therapeutic Approaches in MelanomaCatriona A Ford, Dana Koludrovic, Patricia P Centeno, et al.Nature Medicine|March 29, 2019
Publisher Correction: Protective autophagy elicited by RAF→MEK→ERK inhibition suggests a treatment strategy for RAS-driven cancersConan G Kinsey, Soledad A Camolotto, Amelie M Boespflug, et al.Nature Medicine|March 6, 2019
Protective autophagy elicited by RAF→MEK→ERK inhibition suggests a treatment strategy for RAS-driven cancersConan G Kinsey, Soledad A Camolotto, Amelie M Boespflug, et al.Biorxiv : the Preprint Server for Biology|December 25, 2025
Genetic Drivers of Sensitivity or Resistance to RAS(ON) Multi-Selective Inhibitors in NRAS-Mutated MelanomaMona Foth, Wontak Kim, Kayla T O'Toole, et al.Cancer Cell|June 7, 2016
CXCR2 Inhibition Profoundly Suppresses Metastases and Augments Immunotherapy in Pancreatic Ductal AdenocarcinomaColin W Steele, Saadia A Karim, Joshua D G Leach, et al.Cancer Research|May 22, 2026
Genetic Drivers of Sensitivity or Resistance to RAS(ON) Multi-Selective Inhibitors in NRAS-Mutated MelanomaMona Foth, Wontak Kim, Kayla O'Toole, et al.Pageof 2