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Experimental Neurology
|
December 28, 2011
Protein aggregates in Huntington's disease
Montserrat Arrasate, Steven Finkbeiner
Proceedings of the National Academy of Sciences of the United States of America
|
March 2, 2005
Automated microscope system for determining factors that predict neuronal fate
Montserrat Arrasate, Steven Finkbeiner
Methods in Molecular Biology (Clifton, N.J.)
|
June 18, 2004
Using antibodies to analyze polyglutamine stretches
Elizabeth Brooks, Montserrat Arrasate, Kenneth Cheung, et al.
International Journal of Molecular Sciences
|
July 27, 2022
The Role and Therapeutic Potential of the Integrated Stress Response in Amyotrophic Lateral Sclerosis
Elías Marlin, Cristina Viu-Idocin, Montserrat Arrasate, et al.
Neuroreport
|
April 4, 2002
A two-hybrid screening of human Tau protein: interactions with Alu-derived domain
Janet Hoenicka, Montserrat Arrasate, Justo Garcia de Yebenes, et al.
Nature
|
October 16, 2004
Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death
Montserrat Arrasate, Siddhartha Mitra, Erik S Schweitzer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
August 6, 2010
Quantitative relationships between huntingtin levels, polyglutamine length, inclusion body formation, and neuronal death provide novel insight into huntington's disease molecular pathogenesis
Jason Miller, Montserrat Arrasate, Benjamin A Shaby, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
September 14, 2010
A small-molecule scaffold induces autophagy in primary neurons and protects against toxicity in a Huntington disease model
Andrey S Tsvetkov, Jason Miller, Montserrat Arrasate, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
September 14, 2017
E46K α-synuclein pathological mutation causes cell-autonomous toxicity without altering protein turnover or aggregation
Ignacio Íñigo-Marco, Miguel Valencia, Laura Larrea, et al.
Nature Chemical Biology
|
July 23, 2013
Proteostasis of polyglutamine varies among neurons and predicts neurodegeneration
Andrey S Tsvetkov, Montserrat Arrasate, Sami Barmada, et al.
Page
of 2
Search research articles
Search
Showing results (1-10 of 19) with videos related to
Sort By:
Page
of 2
Experimental Neurology
|
December 28, 2011
Protein aggregates in Huntington's disease
Montserrat Arrasate, Steven Finkbeiner
Proceedings of the National Academy of Sciences of the United States of America
|
March 2, 2005
Automated microscope system for determining factors that predict neuronal fate
Montserrat Arrasate, Steven Finkbeiner
Methods in Molecular Biology (Clifton, N.J.)
|
June 18, 2004
Using antibodies to analyze polyglutamine stretches
Elizabeth Brooks, Montserrat Arrasate, Kenneth Cheung, et al.
International Journal of Molecular Sciences
|
July 27, 2022
The Role and Therapeutic Potential of the Integrated Stress Response in Amyotrophic Lateral Sclerosis
Elías Marlin, Cristina Viu-Idocin, Montserrat Arrasate, et al.
Neuroreport
|
April 4, 2002
A two-hybrid screening of human Tau protein: interactions with Alu-derived domain
Janet Hoenicka, Montserrat Arrasate, Justo Garcia de Yebenes, et al.
Nature
|
October 16, 2004
Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death
Montserrat Arrasate, Siddhartha Mitra, Erik S Schweitzer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
August 6, 2010
Quantitative relationships between huntingtin levels, polyglutamine length, inclusion body formation, and neuronal death provide novel insight into huntington's disease molecular pathogenesis
Jason Miller, Montserrat Arrasate, Benjamin A Shaby, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
September 14, 2010
A small-molecule scaffold induces autophagy in primary neurons and protects against toxicity in a Huntington disease model
Andrey S Tsvetkov, Jason Miller, Montserrat Arrasate, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
September 14, 2017
E46K α-synuclein pathological mutation causes cell-autonomous toxicity without altering protein turnover or aggregation
Ignacio Íñigo-Marco, Miguel Valencia, Laura Larrea, et al.
Nature Chemical Biology
|
July 23, 2013
Proteostasis of polyglutamine varies among neurons and predicts neurodegeneration
Andrey S Tsvetkov, Montserrat Arrasate, Sami Barmada, et al.
Page
of 2