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Experimental Neurology|December 28, 2011
Protein aggregates in Huntington's diseaseMontserrat Arrasate, Steven FinkbeinerProceedings of the National Academy of Sciences of the United States of America|March 2, 2005
Automated microscope system for determining factors that predict neuronal fateMontserrat Arrasate, Steven FinkbeinerNature|October 16, 2004
Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal deathMontserrat Arrasate, Siddhartha Mitra, Erik S Schweitzer, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 6, 2010
Quantitative relationships between huntingtin levels, polyglutamine length, inclusion body formation, and neuronal death provide novel insight into huntington's disease molecular pathogenesisJason Miller, Montserrat Arrasate, Benjamin A Shaby, et al.Proceedings of the National Academy of Sciences of the United States of America|September 14, 2010
A small-molecule scaffold induces autophagy in primary neurons and protects against toxicity in a Huntington disease modelAndrey S Tsvetkov, Jason Miller, Montserrat Arrasate, et al.Nature Chemical Biology|July 23, 2013
Proteostasis of polyglutamine varies among neurons and predicts neurodegenerationAndrey S Tsvetkov, Montserrat Arrasate, Sami Barmada, et al.Nature Medicine|November 6, 2010
Bridging the Valley of Death of therapeutics for neurodegenerationSteven FinkbeinerCold Spring Harbor Perspectives in Biology|April 3, 2019
The Autophagy Lysosomal Pathway and NeurodegenerationSteven FinkbeinerNeurobiology of Disease|September 25, 2020
Functional genomics, genetic risk profiling and cell phenotypes in neurodegenerative diseaseSteven FinkbeinerPageof 14