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Antioxidants (Basel, Switzerland)|March 6, 2021
Mitochondrial Oxidative and Nitrosative Stress as a Therapeutic Target in DiseasesMontserrat Marí, Anna ColellLiver International : Official Journal of the International Association for the Study of the Liver|April 4, 2007
Sphingolipid signalling and liver diseasesMontserrat Marí, José C Fernández-ChecaFree Radical Biology & Medicine|January 5, 2002
Toxicity by pyruvate in HepG2 cells depleted of glutathione: role of mitochondriaMontserrat Marí, Jingxiang Bai, Arthur I CederbaumHepatology (Baltimore, Md.)|December 25, 2002
Cytochrome P450 2E1 responsiveness in the promoter of glutamate-cysteine ligase catalytic subunitNatalia Nieto, Montserrat Marí, Arthur I CederbaumThe Journal of Pharmacology and Experimental Therapeutics|March 22, 2002
Adenovirus-mediated overexpression of catalase in the cytosolic or mitochondrial compartment protects against toxicity caused by glutathione depletion in HepG2 cells expressing CYP2E1Montserrat Marí, Jingxiang Bai, Arthur I CederbaumInternational Journal of Molecular Sciences|June 4, 2020
Relevance of SIRT1-NF-κB Axis as Therapeutic Target to Ameliorate Inflammation in Liver DiseaseEstefanía de Gregorio, Anna Colell, Albert Morales, et al.Seminars in Liver Disease|February 23, 2024
GAS6/TAM Axis as Therapeutic Target in Liver DiseasesAnna Tutusaus, Albert Morales, Pablo García de Frutos, et al.Hepatology (Baltimore, Md.)|January 1, 2009
Cathepsins B and D drive hepatic stellate cell proliferation and promote their fibrogenic potentialAnna Moles, Núria Tarrats, José C Fernández-Checa, et al.The Journal of Biological Chemistry|November 22, 2011
Cathepsin B overexpression due to acid sphingomyelinase ablation promotes liver fibrosis in Niemann-Pick diseaseAnna Moles, Núria Tarrats, José C Fernández-Checa, et al.Current Pharmaceutical Design|September 22, 2011
Metabolic therapy: lessons from liver diseasesCarmen Garcia-Ruiz, Montserrat Marí, Anna Colell, et al.Pageof 6