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Oncotarget|April 19, 2014
Pathways and therapeutic targets in melanomaEmma Shtivelman, Michael Q A Davies, Patrick Hwu, et al.Plos One|May 3, 2011
p53 hypersensitivity is the predominant mechanism of the unique responsiveness of testicular germ cell tumor (TGCT) cells to cisplatinMatthias Gutekunst, Moshe Oren, Andrea Weilbacher, et al.The EMBO Journal|July 12, 2002
Tyrosine phosphorylation of Mdm2 by c-Abl: implications for p53 regulationZehavit Goldberg, Ronit Vogt Sionov, Michael Berger, et al.Journal of Cell Science|August 18, 2012
Mutant p53R273H attenuates the expression of phase 2 detoxifying enzymes and promotes the survival of cells with high levels of reactive oxygen speciesEyal Kalo, Ira Kogan-Sakin, Hilla Solomon, et al.Nature Communications|May 18, 2022
Visual barcodes for clonal-multiplexing of live microscopy-based assaysTom Kaufman, Erez Nitzan, Nir Firestein, et al.Cancer Research|June 3, 2005
Transcriptional programs following genetic alterations in p53, INK4A, and H-Ras genes along defined stages of malignant transformationMichael Milyavsky, Yuval Tabach, Igor Shats, et al.Cancer Research|January 11, 2013
Cisplatin hypersensitivity of testicular germ cell tumors is determined by high constitutive Noxa levels mediated by Oct-4Matthias Gutekunst, Thomas Mueller, Andrea Weilbacher, et al.EMBO Reports|July 4, 2009
CDK9 directs H2B monoubiquitination and controls replication-dependent histone mRNA 3'-end processingJudith Pirngruber, Andrei Shchebet, Lisa Schreiber, et al.Apoptosis : an International Journal on Programmed Cell Death|April 29, 2024
ARTS and small-molecule ARTS mimetics upregulate p53 levels by promoting the degradation of XIAPRuqaia Abbas, Oliver Hartmann, Dorin Theodora Asiss, et al.Genes & Development|June 14, 2017
p53 is essential for DNA methylation homeostasis in naïve embryonic stem cells, and its loss promotes clonal heterogeneityAyala Tovy, Adam Spiro, Ryan McCarthy, et al.Pageof 27