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Genes & Development|March 26, 2016
The LATS2 tumor suppressor inhibits SREBP and suppresses hepatic cholesterol accumulationYael Aylon, Anat Gershoni, Ron Rotkopf, et al.Life Science Alliance|November 21, 2018
LATS1 and LATS2 suppress breast cancer progression by maintaining cell identity and metabolic stateNoa Furth, Ioannis S Pateras, Ron Rotkopf, et al.Molecular Oncology|September 16, 2023
TAZ facilitates breast tumor growth by promoting an immune-suppressive tumor microenvironmentAnat Gershoni, Ori Hassin, Nishanth Belugali Nataraj, et al.Proceedings of the National Academy of Sciences of the United States of America|January 9, 2003
Nitric oxide-induced cellular stress and p53 activation in chronic inflammationLorne J Hofseth, Shin'ichi Saito, S Perwez Hussain, et al.Proceedings of the National Academy of Sciences of the United States of America|February 27, 2004
Siah-1b is a direct transcriptional target of p53: identification of the functional p53 responsive element in the siah-1b promoterGiusy Fiucci, Séverine Beaucourt, Dominique Duflaut, et al.Proceedings of the National Academy of Sciences of the United States of America|October 29, 2025
p53 regulates the expression of histone modifiers to restrict stemness and maintain differentiated luminal identity in breast cancerYael Aylon, Noa Furth, Anna C Pirona, et al.Cancer Research|December 20, 2018
Early Loss of Histone H2B Monoubiquitylation Alters Chromatin Accessibility and Activates Key Immune Pathways That Facilitate Progression of Ovarian CancerJagmohan Hooda, Marián Novak, Matthew P Salomon, et al.Cell Death & Disease|April 24, 2024
ATF3 characterizes aggressive drug-tolerant persister cells in HGSOCKathrin Böpple, Yaara Oren, Whitney S Henry, et al.Nature|February 19, 2011
CKIα ablation highlights a critical role for p53 in invasiveness controlEla Elyada, Ariel Pribluda, Robert E Goldstein, et al.Oncogene|January 31, 2018
PAX8 activates a p53-p21-dependent pro-proliferative effect in high grade serous ovarian carcinomaDima Ghannam-Shahbari, Eyal Jacob, Reli Rachel Kakun, et al.Pageof 27