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Nucleic Acids Research|October 17, 2017
Epigenetic mechanisms underlie the crosstalk between growth factors and a steroid hormoneYehoshua Enuka, Morris E Feldman, Animesh Chowdhury, et al.Genes & Development|October 4, 2008
The histone H2B-specific ubiquitin ligase RNF20/hBRE1 acts as a putative tumor suppressor through selective regulation of gene expressionEfrat Shema, Itay Tirosh, Yael Aylon, et al.Cancer Cell|July 30, 2013
A senescence-inflammatory switch from cancer-inhibitory to cancer-promoting mechanismAriel Pribluda, Ela Elyada, Zoltan Wiener, et al.Nature Communications|May 19, 2022
Different hotspot p53 mutants exert distinct phenotypes and predict outcome of colorectal cancer patientsOri Hassin, Nishanth Belugali Nataraj, Michal Shreberk-Shaked, et al.Proceedings of the National Academy of Sciences of the United States of America|April 19, 2022
Cross-talk between mutant p53 and p62/SQSTM1 augments cancer cell migration by promoting the degradation of cell adhesion proteinsSaptaparna Mukherjee, Martino Maddalena, YiQing Lü, et al.Cell Reports|May 26, 2021
PD-L1 recruits phospholipase C and enhances tumorigenicity of lung tumors harboring mutant forms of EGFRSoma Ghosh, Nishanth Belugali Nataraj, Ashish Noronha, et al.Proceedings of the National Academy of Sciences of the United States of America|December 21, 2023
p53 deficient breast cancer cells reprogram preadipocytes toward tumor-protective immunomodulatory cellsOri Hassin, Miriam Sernik, Adi Seligman, et al.Nature Cardiovascular Research|August 28, 2024
Egr1 regulates regenerative senescence and cardiac repairLingling Zhang, Jacob Elkahal, Tianzhen Wang, et al.Cell Stem Cell|April 29, 2021
Principles of signaling pathway modulation for enhancing human naive pluripotency inductionJonathan Bayerl, Muneef Ayyash, Tom Shani, et al.Nature Communications|January 5, 2023
Germline TP53 mutations undergo copy number gain years prior to tumor diagnosisNicholas Light, Mehdi Layeghifard, Ayush Attery, et al.Pageof 27