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Moshe Oren

Showing results (91-100 of 172) with videos related to

Pageof 18
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Oncotarget|April 19, 2014
Pathways and therapeutic targets in melanomaEmma Shtivelman, Michael Q A Davies, Patrick Hwu, et al.
Plos One|May 3, 2011
p53 hypersensitivity is the predominant mechanism of the unique responsiveness of testicular germ cell tumor (TGCT) cells to cisplatinMatthias Gutekunst, Moshe Oren, Andrea Weilbacher, et al.
Omics : a Journal of Integrative Biology|February 22, 2011
ChIP-on-chip analysis of in vivo mutant p53 binding to selected gene promotersStefania Dell'Orso, Giulia Fontemaggi, Perry Stambolsky, et al.
Oncogene|August 29, 2003
Mutant p53 gain of function: repression of CD95(Fas/APO-1) gene expression by tumor-associated p53 mutantsAmir Zalcenstein, Perry Stambolsky, Lilach Weisz, et al.
Cell Death and Differentiation|October 15, 2016
TM7SF3, a novel p53-regulated homeostatic factor, attenuates cellular stress and the subsequent induction of the unfolded protein responseRoi Isaac, Ido Goldstein, Noa Furth, et al.
The EMBO Journal|July 12, 2002
Tyrosine phosphorylation of Mdm2 by c-Abl: implications for p53 regulationZehavit Goldberg, Ronit Vogt Sionov, Michael Berger, et al.
Genes & Development|June 14, 2017
p53 is essential for DNA methylation homeostasis in naïve embryonic stem cells, and its loss promotes clonal heterogeneityAyala Tovy, Adam Spiro, Ryan McCarthy, et al.
Genes & Development|October 3, 2018
Corrigendum: p53 is essential for DNA methylation homeostasis in naïve embryonic stem cells, and its loss promotes clonal heterogeneityAyala Tovy, Adam Spiro, Ryan McCarthy, et al.
Cancer Research|January 11, 2013
Cisplatin hypersensitivity of testicular germ cell tumors is determined by high constitutive Noxa levels mediated by Oct-4Matthias Gutekunst, Thomas Mueller, Andrea Weilbacher, et al.
EMBO Reports|July 4, 2009
CDK9 directs H2B monoubiquitination and controls replication-dependent histone mRNA 3'-end processingJudith Pirngruber, Andrei Shchebet, Lisa Schreiber, et al.
Pageof 18

Showing results (91-100 of 172) with videos related to

Sort By:
Pageof 18
Oncotarget|April 19, 2014
Pathways and therapeutic targets in melanomaEmma Shtivelman, Michael Q A Davies, Patrick Hwu, et al.
Plos One|May 3, 2011
p53 hypersensitivity is the predominant mechanism of the unique responsiveness of testicular germ cell tumor (TGCT) cells to cisplatinMatthias Gutekunst, Moshe Oren, Andrea Weilbacher, et al.
Omics : a Journal of Integrative Biology|February 22, 2011
ChIP-on-chip analysis of in vivo mutant p53 binding to selected gene promotersStefania Dell'Orso, Giulia Fontemaggi, Perry Stambolsky, et al.
Oncogene|August 29, 2003
Mutant p53 gain of function: repression of CD95(Fas/APO-1) gene expression by tumor-associated p53 mutantsAmir Zalcenstein, Perry Stambolsky, Lilach Weisz, et al.
Cell Death and Differentiation|October 15, 2016
TM7SF3, a novel p53-regulated homeostatic factor, attenuates cellular stress and the subsequent induction of the unfolded protein responseRoi Isaac, Ido Goldstein, Noa Furth, et al.
The EMBO Journal|July 12, 2002
Tyrosine phosphorylation of Mdm2 by c-Abl: implications for p53 regulationZehavit Goldberg, Ronit Vogt Sionov, Michael Berger, et al.
Genes & Development|June 14, 2017
p53 is essential for DNA methylation homeostasis in naïve embryonic stem cells, and its loss promotes clonal heterogeneityAyala Tovy, Adam Spiro, Ryan McCarthy, et al.
Genes & Development|October 3, 2018
Corrigendum: p53 is essential for DNA methylation homeostasis in naïve embryonic stem cells, and its loss promotes clonal heterogeneityAyala Tovy, Adam Spiro, Ryan McCarthy, et al.
Cancer Research|January 11, 2013
Cisplatin hypersensitivity of testicular germ cell tumors is determined by high constitutive Noxa levels mediated by Oct-4Matthias Gutekunst, Thomas Mueller, Andrea Weilbacher, et al.
EMBO Reports|July 4, 2009
CDK9 directs H2B monoubiquitination and controls replication-dependent histone mRNA 3'-end processingJudith Pirngruber, Andrei Shchebet, Lisa Schreiber, et al.
Pageof 18