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Molecular Cell
|
May 21, 2011
Two phases of mitogenic signaling unveil roles for p53 and EGR1 in elimination of inconsistent growth signals
Yaara Zwang, Aldema Sas-Chen, Yotam Drier, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
February 28, 2003
The p53-inducible TSAP6 gene product regulates apoptosis and the cell cycle and interacts with Nix and the Myt1 kinase
Brent J Passer, Vanessa Nancy-Portebois, Nathalie Amzallag, et al.
Cancer Cell
|
May 18, 2013
Mutant p53 prolongs NF-κB activation and promotes chronic inflammation and inflammation-associated colorectal cancer
Tomer Cooks, Ioannis S Pateras, Ohad Tarcic, et al.
Oncogene
|
January 19, 2018
Mutant p53 gain of function underlies high expression levels of colorectal cancer stem cells markers
Hilla Solomon, Nathan Dinowitz, Ioannis S Pateras, et al.
Nature Communications
|
January 10, 2023
Author Correction: Breast cancer plasticity is restricted by a LATS1-NCOR1 repressive axis
Yael Aylon, Noa Furth, Giuseppe Mallel, et al.
Nature Communications
|
November 28, 2022
Breast cancer plasticity is restricted by a LATS1-NCOR1 repressive axis
Yael Aylon, Noa Furth, Giuseppe Mallel, et al.
Gastroenterology
|
April 12, 2016
Genomic Alterations Observed in Colitis-Associated Cancers Are Distinct From Those Found in Sporadic Colorectal Cancers and Vary by Type of Inflammatory Bowel Disease
Rona Yaeger, Manish A Shah, Vincent A Miller, et al.
Blood
|
April 15, 2018
TRRAP is essential for regulating the accumulation of mutant and wild-type p53 in lymphoma
Alexander Jethwa, Mikołaj Słabicki, Jennifer Hüllein, et al.
Molecular Systems Biology
|
November 27, 2008
p53-Repressed miRNAs are involved with E2F in a feed-forward loop promoting proliferation
Ran Brosh, Reut Shalgi, Atar Liran, et al.
Genes & Development
|
October 21, 2017
Corrigendum: The histone H2B-specific ubiquitin ligase RNF20/hBRE1 acts as a putative tumor suppressor through selective regulation of gene expression
Efrat Shema, Itay Tirosh, Yael Aylon, et al.
Page
of 18
Search research articles
Search
Showing results (141-150 of 172) with videos related to
Sort By:
Page
of 18
Molecular Cell
|
May 21, 2011
Two phases of mitogenic signaling unveil roles for p53 and EGR1 in elimination of inconsistent growth signals
Yaara Zwang, Aldema Sas-Chen, Yotam Drier, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
February 28, 2003
The p53-inducible TSAP6 gene product regulates apoptosis and the cell cycle and interacts with Nix and the Myt1 kinase
Brent J Passer, Vanessa Nancy-Portebois, Nathalie Amzallag, et al.
Cancer Cell
|
May 18, 2013
Mutant p53 prolongs NF-κB activation and promotes chronic inflammation and inflammation-associated colorectal cancer
Tomer Cooks, Ioannis S Pateras, Ohad Tarcic, et al.
Oncogene
|
January 19, 2018
Mutant p53 gain of function underlies high expression levels of colorectal cancer stem cells markers
Hilla Solomon, Nathan Dinowitz, Ioannis S Pateras, et al.
Nature Communications
|
January 10, 2023
Author Correction: Breast cancer plasticity is restricted by a LATS1-NCOR1 repressive axis
Yael Aylon, Noa Furth, Giuseppe Mallel, et al.
Nature Communications
|
November 28, 2022
Breast cancer plasticity is restricted by a LATS1-NCOR1 repressive axis
Yael Aylon, Noa Furth, Giuseppe Mallel, et al.
Gastroenterology
|
April 12, 2016
Genomic Alterations Observed in Colitis-Associated Cancers Are Distinct From Those Found in Sporadic Colorectal Cancers and Vary by Type of Inflammatory Bowel Disease
Rona Yaeger, Manish A Shah, Vincent A Miller, et al.
Blood
|
April 15, 2018
TRRAP is essential for regulating the accumulation of mutant and wild-type p53 in lymphoma
Alexander Jethwa, Mikołaj Słabicki, Jennifer Hüllein, et al.
Molecular Systems Biology
|
November 27, 2008
p53-Repressed miRNAs are involved with E2F in a feed-forward loop promoting proliferation
Ran Brosh, Reut Shalgi, Atar Liran, et al.
Genes & Development
|
October 21, 2017
Corrigendum: The histone H2B-specific ubiquitin ligase RNF20/hBRE1 acts as a putative tumor suppressor through selective regulation of gene expression
Efrat Shema, Itay Tirosh, Yael Aylon, et al.
Page
of 18