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Biological Psychiatry|November 27, 2021
A Perspective on the Potential Involvement of Impaired Proteostasis in Neuropsychiatric DisordersKelvin K Hui, Ryo Endo, Akira Sawa, et al.The Journal of Biological Chemistry|November 24, 2006
Biochemical and functional analysis of the assembly of full-length Sup35p and its prion-forming domainJoanna Krzewska, Motomasa Tanaka, Steven G Burston, et al.Cell|April 12, 2005
Mechanism of cross-species prion transmission: an infectious conformation compatible with two highly divergent yeast prion proteinsMotomasa Tanaka, Peter Chien, Koji Yonekura, et al.Biomolecules|November 6, 2019
Translation from the Ribosome to the Clinic: Implication in Neurological Disorders and New Perspectives from Recent AdvancesKelvin K Hui, Yi-Kai Chen, Ryo Endo, et al.Frontiers in Molecular Neuroscience|July 25, 2022
How Staying Negative Is Good for the (Adult) Brain: Maintaining Chloride Homeostasis and the GABA-Shift in Neurological DisordersKelvin K Hui, Thomas E Chater, Yukiko Goda, et al.Nature|March 19, 2004
Conformational variations in an infectious protein determine prion strain differencesMotomasa Tanaka, Peter Chien, Nariman Naber, et al.Nature|July 1, 2006
The physical basis of how prion conformations determine strain phenotypesMotomasa Tanaka, Sean R Collins, Brandon H Toyama, et al.Molecular Cell|April 8, 2004
Eukaryotic proteasomes cannot digest polyglutamine sequences and release them during degradation of polyglutamine-containing proteinsPrasanna Venkatraman, Ronald Wetzel, Motomasa Tanaka, et al.Nature Chemical Biology|January 19, 2010
Differences in prion strain conformations result from non-native interactions in a nucleusYumiko Ohhashi, Kazuki Ito, Brandon H Toyama, et al.Genes to Cells : Devoted to Molecular & Cellular Mechanisms|April 7, 2011
Aging causes distinct characteristics of polyglutamine amyloids in vivoAyako Tonoki, Erina Kuranaga, Natsuki Ito, et al.Pageof 4