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Biorxiv : the Preprint Server for Biology|September 10, 2024
Break-induced replication drives large-scale genomic amplifications in cancer cellsMouadh Benamar, Rebeka Eki, Kang-Ping Du, et al.Nucleic Acids Research|October 17, 2020
A robust CRISPR-Cas9-based fluorescent reporter assay for the detection and quantification of DNA double-strand break repairRebeka Eki, Jane She, Mahmut Parlak, et al.Molecular Cancer Therapeutics|August 26, 2017
The Neddylation Inhibitor Pevonedistat (MLN4924) Suppresses and Radiosensitizes Head and Neck Squamous Carcinoma Cells and TumorsVanessa Vanderdys, Amir Allak, Fadila Guessous, et al.Nucleic Acids Research|October 31, 2021
MicroDNA levels are dependent on MMEJ, repressed by c-NHEJ pathway, and stimulated by DNA damageTeressa Paulsen, Pumoli Malapati, Yoshiyuki Shibata, et al.Ebiomedicine|June 23, 2016
Inactivation of the CRL4-CDT2-SET8/p21 ubiquitylation and degradation axis underlies the therapeutic efficacy of pevonedistat in melanomaMouadh Benamar, Fadila Guessous, Kangping Du, et al.The Journal of Clinical Investigation|May 17, 2024
Somatic structural variants drive distinct modes of oncogenesis in melanomaJake R Conway, Riaz Gillani, Jett Crowdis, et al.Cancer Research|May 31, 2018
Combined c-Met/Trk Inhibition Overcomes Resistance to CDK4/6 Inhibitors in GlioblastomaInan Olmez, Ying Zhang, Laryssa Manigat, et al.International Journal of Radiation Oncology, Biology, Physics|July 19, 2014
Targeting nucleophosmin 1 represents a rational strategy for radiation sensitizationKonjeti R Sekhar, Mouadh Benamar, Amudhan Venkateswaran, et al.Annals of Surgery|August 20, 2019
The Barrier Molecules Junction Plakoglobin, Filaggrin, and Dystonin Play Roles in Melanoma Growth and AngiogenesisKatie M Leick, Anthony B Rodriguez, Marit M Melssen, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|August 18, 2017
Combined CDK4/6 and mTOR Inhibition Is Synergistic against Glioblastoma via Multiple MechanismsInan Olmez, Breanna Brenneman, Aizhen Xiao, et al.Pageof 2