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Cancer Letters|June 14, 2005
Bloom syndrome, genomic instability and cancer: the SOS-like hypothesisMounira Amor-Guéret
Methods in Molecular Biology (Clifton, N.J.)|August 26, 2020
Identification and Analysis of Different Types of UFBsSimon Gemble, Mounira Amor-Guéret
Oncogene|April 18, 2002
Bloom's syndrome protein response to ultraviolet-C radiation and hydroxyurea-mediated DNA synthesis inhibitionMouna Ababou, Virginie Dumaire, Yann Lécluse, et al.
Cell Cycle (Georgetown, Tex.)|November 14, 2002
Cleavage of BLM and sensitivity of Bloom's syndrome cells to hydroxurea and UV-C radiationMouna Ababou, Virginie Dumaire, Yann Lécluse, et al.
Trends in Biochemical Sciences|February 19, 2014
DNA damage: RNA-binding proteins protect from near and farMartin Dutertre, Sarah Lambert, Aura Carreira, et al.
Cancer Research|December 18, 2004
A major role for mitotic CDC2 kinase inactivation in the establishment of the mitotic DNA damage checkpointEmilie Bayart, Olga Grigorieva, Serge Leibovitch, et al.
Cell Cycle (Georgetown, Tex.)|May 3, 2017
Cytidine deaminase deficiency impairs sister chromatid disjunction by decreasing PARP-1 activitySimon Gemble, Géraldine Buhagiar-Labarchède, Rosine Onclercq-Delic, et al.
Nucleic Acids Research|October 17, 2017
PARP2 controls double-strand break repair pathway choice by limiting 53BP1 accumulation at DNA damage sites and promoting end-resectionAlexis Fouquin, Josée Guirouilh-Barbat, Bernard Lopez, et al.
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