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Life Science Alliance|August 3, 2023
PRMT5 triggers glucocorticoid-induced cell migration in triple-negative breast cancerLara Malik Noureddine, Julien Ablain, Ausra Surmieliova-Garnès, et al.
Journal of Inflammation Research|May 28, 2021
A Gender-Dependent Molecular Switch of Inflammation via MyD88/Estrogen Receptor-Alpha InteractionRana El Sabeh, Mélanie Bonnet, Katy Le Corf, et al.
Oncogene|December 16, 2020
Men1 disruption in Nkx3.1-deficient mice results in AR<sup>low</sup>/CD44<sup>+</sup> microinvasive carcinoma development with the dysregulated AR pathwayRomain Teinturier, Yakun Luo, Myriam Decaussin-Petrucci, et al.
Breast Cancer Research : BCR|May 22, 2021
Analysis of genomic and non-genomic signaling of estrogen receptor in PDX models of breast cancer treated with a combination of the PI3K inhibitor alpelisib (BYL719) and fulvestrantJulien Jacquemetton, Loay Kassem, Coralie Poulard, et al.
Cancers|January 21, 2022
PRMT1 Regulates EGFR and Wnt Signaling Pathways and Is a Promising Target for Combinatorial Treatment of Breast CancerSamyuktha Suresh, Solène Huard, Amélie Brisson, et al.
Science Advances|March 15, 2023
A hotspot for posttranslational modifications on the androgen receptor dimer interface drives pathology and anti-androgen resistanceAndrea Alegre-Martí, Alba Jiménez-Panizo, Adrián Martínez-Tébar, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|July 10, 2020
Mutation of Arginine 264 on ERα (Estrogen Receptor Alpha) Selectively Abrogates the Rapid Signaling of Estradiol in the Endothelium Without Altering FertilityMarine Adlanmerini, Chanaelle Fébrissy, Rana Zahreddine, et al.
Cell Discovery|January 31, 2024
Cytoskeleton remodeling induced by SMYD2 methyltransferase drives breast cancer metastasisAlexandre G Casanova, Gael S Roth, Simone Hausmann, et al.
Nature Communications|August 15, 2020
PLK1 inhibition exhibits strong anti-tumoral activity in CCND1-driven breast cancer metastases with acquired palbociclib resistanceElodie Montaudon, Joanna Nikitorowicz-Buniak, Laura Sourd, et al.
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