Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Murray Clarke

Showing results (1-10 of 9) with videos related to

Pageof 1
Sort By:
American Journal of Nephrology|December 13, 2006
The emerging role of vascular smooth muscle cell apoptosis in atherosclerosis and plaque stabilityMurray Clarke, Martin Bennett
Cell Cycle (Georgetown, Tex.)|October 3, 2006
Defining the role of vascular smooth muscle cell apoptosis in atherosclerosisMurray Clarke, Martin Bennett
Heart (British Cardiac Society)|March 21, 2006
Cell death in the cardiovascular systemMurray Clarke, Martin Bennett, Trevor Littlewood
Vascular Pharmacology|February 7, 2012
Signalling from dead cells drives inflammation and vessel remodellingMartin Bennett, Haixiang Yu, Murray Clarke
Apoptosis : an International Journal on Programmed Cell Death|July 7, 2020
Cytokine regulation of apoptosis-induced apoptosis and apoptosis-induced cell proliferation in vascular smooth muscle cellsDimitra Aravani, Kirsty Foote, Nichola Figg, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 5, 2011
Bone marrow-derived smooth muscle-like cells are infrequent in advanced primary atherosclerotic plaques but promote atherosclerosisHaixiang Yu, Victoria Stoneman, Murray Clarke, et al.
Circulation|September 30, 2015
Vascular Smooth Muscle Cell Senescence Promotes Atherosclerosis and Features of Plaque VulnerabilityJulie Wang, Anna K Uryga, Johannes Reinhold, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 18, 2011
Leukocyte telomere length is associated with high-risk plaques on virtual histology intravascular ultrasound and increased proinflammatory activityPatrick A Calvert, Tze-Vun Liew, Isabelle Gorenne, et al.
The Journal of Clinical Investigation|September 12, 2023
PLK1 inhibition dampens NLRP3 inflammasome-elicited response in inflammatory disease modelsMarta Baldrighi, Christian Doreth, Yang Li, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
American Journal of Nephrology|December 13, 2006
The emerging role of vascular smooth muscle cell apoptosis in atherosclerosis and plaque stabilityMurray Clarke, Martin Bennett
Cell Cycle (Georgetown, Tex.)|October 3, 2006
Defining the role of vascular smooth muscle cell apoptosis in atherosclerosisMurray Clarke, Martin Bennett
Heart (British Cardiac Society)|March 21, 2006
Cell death in the cardiovascular systemMurray Clarke, Martin Bennett, Trevor Littlewood
Vascular Pharmacology|February 7, 2012
Signalling from dead cells drives inflammation and vessel remodellingMartin Bennett, Haixiang Yu, Murray Clarke
Apoptosis : an International Journal on Programmed Cell Death|July 7, 2020
Cytokine regulation of apoptosis-induced apoptosis and apoptosis-induced cell proliferation in vascular smooth muscle cellsDimitra Aravani, Kirsty Foote, Nichola Figg, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 5, 2011
Bone marrow-derived smooth muscle-like cells are infrequent in advanced primary atherosclerotic plaques but promote atherosclerosisHaixiang Yu, Victoria Stoneman, Murray Clarke, et al.
Circulation|September 30, 2015
Vascular Smooth Muscle Cell Senescence Promotes Atherosclerosis and Features of Plaque VulnerabilityJulie Wang, Anna K Uryga, Johannes Reinhold, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 18, 2011
Leukocyte telomere length is associated with high-risk plaques on virtual histology intravascular ultrasound and increased proinflammatory activityPatrick A Calvert, Tze-Vun Liew, Isabelle Gorenne, et al.
The Journal of Clinical Investigation|September 12, 2023
PLK1 inhibition dampens NLRP3 inflammasome-elicited response in inflammatory disease modelsMarta Baldrighi, Christian Doreth, Yang Li, et al.
Pageof 1