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Small Gtpases|May 11, 2017
Evidence for the subsynaptic zone as a preferential site for CHRN recycling at neuromuscular junctionsFranziska Wild, Muzamil Majid Khan, Rüdiger RudolfCells|May 1, 2019
Motor Endplate-Anatomical, Functional, and Molecular Concepts in the Historical PerspectiveRüdiger Rudolf, Muzamil Majid Khan, Veit WitzemannFrontiers in Bioscience (Landmark Edition)|April 22, 2016
The impact of autophagy on peripheral synapses in health and diseaseRudiger Rudolf, Muzamil Majid Khan, Franziska Wild, et al.Autophagy|October 8, 2016
Progress of endocytic CHRN to autophagic degradation is regulated by RAB5-GTPase and T145 phosphorylation of SH3GLB1 at mouse neuromuscular junctions in vivoFranziska Wild, Muzamil Majid Khan, Tatjana Straka, et al.Frontiers in Aging Neuroscience|June 7, 2014
Degeneration of neuromuscular junction in age and dystrophyRüdiger Rudolf, Muzamil Majid Khan, Siegfried Labeit, et al.Journal of Muscle Research and Cell Motility|August 16, 2015
Turnover of acetylcholine receptors at the endplate revisited: novel insights into nerve-dependent behaviorSiegfried Strack, Muzamil Majid Khan, Franziska Wild, et al.The Journal of Cell Biology|July 13, 2022
Regulation of the COPII secretory machinery via focal adhesions and extracellular matrix signalingJuan Jung, Muzamil Majid Khan, Jonathan Landry, et al.The Journal of Cell Biology|July 28, 2026
Spatiotemporal regulation of DNA repair proteins between Golgi and nucleus maintains genome stabilityGeorge Galea, Karolina Kuodyte, Muzamil Majid Khan, et al.Plos One|July 21, 2012
Participation of myosin Va and Pka type I in the regeneration of neuromuscular junctionsIra Verena Röder, Siegfried Strack, Markus Reischl, et al.Human Molecular Genetics|June 16, 2018
GFPT1 deficiency in muscle leads to myasthenia and myopathy in miceYasmin Issop, Denisa Hathazi, Muzamil Majid Khan, et al.Pageof 2