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N A DeLuca

Showing results (11-20 of 46) with videos related to

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Journal of Virology|September 1, 1992
Substitution of a TATA box from a herpes simplex virus late gene in the viral thymidine kinase promoter alters ICP4 inducibility but not temporal expressionA N Imbalzano, N A DeLuca
Journal of Virology|August 8, 1998
The polyserine tract of herpes simplex virus ICP4 is required for normal viral gene expression and growth in murine trigeminal gangliaP A Bates, N A DeLuca
Journal of Virology|September 1, 1995
Relationship between TATA-binding protein and herpes simplex virus type 1 ICP4 DNA-binding sites in complex formation and repression of transcriptionR Kuddus, B Gu, N A DeLuca
Molecular and Cellular Biology|July 1, 1995
Repression of activator-mediated transcription by herpes simplex virus ICP4 via a mechanism involving interactions with the basal transcription factors TATA-binding protein and TFIIBB Gu, R Kuddus, N A DeLuca
Virology|July 31, 1998
Herpes simplex virus type 1 VP26 is not essential for replication in cell culture but influences production of infectious virus in the nervous system of infected miceP Desai, N A DeLuca, S Person
Journal of Virology|April 3, 1998
Persistence and expression of the herpes simplex virus genome in the absence of immediate-early proteinsL A Samaniego, L Neiderhiser, N A DeLuca
Journal of Virology|February 1, 1996
Analysis of phosphorylation sites of herpes simplex virus type 1 ICP4K Xia, N A DeLuca, D M Knipe
Journal of Virology|February 1, 1996
Role of protein kinase A and the serine-rich region of herpes simplex virus type 1 ICP4 in viral replicationK Xia, D M Knipe, N A DeLuca
Journal of Virology|August 1, 1990
trans-dominant inhibition of herpes simplex virus transcriptional regulatory protein ICP4 by heterodimer formationA A Shepard, P Tolentino, N A DeLuca
Journal of Virology|August 1, 1996
Overexpression of the herpes simplex virus type 1 immediate-early regulatory protein, ICP27, is responsible for the aberrant localization of ICP0 and mutant forms of ICP4 in ICP4 mutant virus-infected cellsZ Zhu, N A DeLuca, P A Schaffer
Pageof 5

Showing results (11-20 of 46) with videos related to

Sort By:
Pageof 5
Journal of Virology|September 1, 1992
Substitution of a TATA box from a herpes simplex virus late gene in the viral thymidine kinase promoter alters ICP4 inducibility but not temporal expressionA N Imbalzano, N A DeLuca
Journal of Virology|August 8, 1998
The polyserine tract of herpes simplex virus ICP4 is required for normal viral gene expression and growth in murine trigeminal gangliaP A Bates, N A DeLuca
Journal of Virology|September 1, 1995
Relationship between TATA-binding protein and herpes simplex virus type 1 ICP4 DNA-binding sites in complex formation and repression of transcriptionR Kuddus, B Gu, N A DeLuca
Molecular and Cellular Biology|July 1, 1995
Repression of activator-mediated transcription by herpes simplex virus ICP4 via a mechanism involving interactions with the basal transcription factors TATA-binding protein and TFIIBB Gu, R Kuddus, N A DeLuca
Virology|July 31, 1998
Herpes simplex virus type 1 VP26 is not essential for replication in cell culture but influences production of infectious virus in the nervous system of infected miceP Desai, N A DeLuca, S Person
Journal of Virology|April 3, 1998
Persistence and expression of the herpes simplex virus genome in the absence of immediate-early proteinsL A Samaniego, L Neiderhiser, N A DeLuca
Journal of Virology|February 1, 1996
Analysis of phosphorylation sites of herpes simplex virus type 1 ICP4K Xia, N A DeLuca, D M Knipe
Journal of Virology|February 1, 1996
Role of protein kinase A and the serine-rich region of herpes simplex virus type 1 ICP4 in viral replicationK Xia, D M Knipe, N A DeLuca
Journal of Virology|August 1, 1990
trans-dominant inhibition of herpes simplex virus transcriptional regulatory protein ICP4 by heterodimer formationA A Shepard, P Tolentino, N A DeLuca
Journal of Virology|August 1, 1996
Overexpression of the herpes simplex virus type 1 immediate-early regulatory protein, ICP27, is responsible for the aberrant localization of ICP0 and mutant forms of ICP4 in ICP4 mutant virus-infected cellsZ Zhu, N A DeLuca, P A Schaffer
Pageof 5