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N Abell

Showing results (21-30 of 40) with videos related to

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The Journal of Biological Chemistry|December 18, 1998
Mutations that induce constitutive activation and mutations that impair signal transduction modulate the basal and/or agonist-stimulated internalization of the Lutropin/Choriogonadotropin receptorK S Min, X Liu, J Fabritz, et al.
Cell Reports|March 28, 2019
GALNT3 Maintains the Epithelial State in Trophoblast Stem CellsDeepthi Raghu, Robert J Mobley, Noha A M Shendy, et al.
Frontiers in Cell and Developmental Biology|February 23, 2023
The role of WNT10B in physiology and disease: A 10-year updateRachel S Perkins, Rishika Singh, Amy N Abell, et al.
Nucleic Acids Research|August 2, 2011
Efficiently identifying genome-wide changes with next-generation sequencing dataWeichun Huang, David M Umbach, Nicole Vincent Jordan, et al.
International Journal of Molecular Sciences|September 9, 2022
CYP1B1 Augments the Mesenchymal, Claudin-Low, and Chemoresistant Phenotypes of Triple-Negative Breast Cancer CellsPaul R Hollis, Robert J Mobley, Jyoti Bhuju, et al.
Journal of Cell Science|December 17, 2003
Rac2D57N, a dominant inhibitory Rac2 mutant that inhibits p38 kinase signaling and prevents surface ruffling in bone-marrow-derived macrophagesAmy N Abell, Aimee M DeCathelineau, Scott A Weed, et al.
Molecular and Cellular Biology|March 18, 2009
Trophoblast stem cell maintenance by fibroblast growth factor 4 requires MEKK4 activation of Jun N-terminal kinaseAmy N Abell, Deborah A Granger, Nancy L Johnson, et al.
The Journal of Biological Chemistry|February 26, 2010
Rho kinase inhibition rescues the endothelial cell cerebral cavernous malformation phenotypeAsya L Borikova, Christopher F Dibble, Noah Sciaky, et al.
Human Molecular Genetics|July 23, 2005
CCM1 and CCM2 protein interactions in cell signaling: implications for cerebral cavernous malformations pathogenesisJon S Zawistowski, Lisa Stalheim, Mark T Uhlik, et al.
The Journal of Clinical Endocrinology and Metabolism|July 14, 1998
A unique constitutively activating mutation in third transmembrane helix of luteinizing hormone receptor causes sporadic male gonadotropin-independent precocious pubertyA C Latronico, A N Abell, I J Arnhold, et al.
Pageof 4

Showing results (21-30 of 40) with videos related to

Sort By:
Pageof 4
The Journal of Biological Chemistry|December 18, 1998
Mutations that induce constitutive activation and mutations that impair signal transduction modulate the basal and/or agonist-stimulated internalization of the Lutropin/Choriogonadotropin receptorK S Min, X Liu, J Fabritz, et al.
Cell Reports|March 28, 2019
GALNT3 Maintains the Epithelial State in Trophoblast Stem CellsDeepthi Raghu, Robert J Mobley, Noha A M Shendy, et al.
Frontiers in Cell and Developmental Biology|February 23, 2023
The role of WNT10B in physiology and disease: A 10-year updateRachel S Perkins, Rishika Singh, Amy N Abell, et al.
Nucleic Acids Research|August 2, 2011
Efficiently identifying genome-wide changes with next-generation sequencing dataWeichun Huang, David M Umbach, Nicole Vincent Jordan, et al.
International Journal of Molecular Sciences|September 9, 2022
CYP1B1 Augments the Mesenchymal, Claudin-Low, and Chemoresistant Phenotypes of Triple-Negative Breast Cancer CellsPaul R Hollis, Robert J Mobley, Jyoti Bhuju, et al.
Journal of Cell Science|December 17, 2003
Rac2D57N, a dominant inhibitory Rac2 mutant that inhibits p38 kinase signaling and prevents surface ruffling in bone-marrow-derived macrophagesAmy N Abell, Aimee M DeCathelineau, Scott A Weed, et al.
Molecular and Cellular Biology|March 18, 2009
Trophoblast stem cell maintenance by fibroblast growth factor 4 requires MEKK4 activation of Jun N-terminal kinaseAmy N Abell, Deborah A Granger, Nancy L Johnson, et al.
The Journal of Biological Chemistry|February 26, 2010
Rho kinase inhibition rescues the endothelial cell cerebral cavernous malformation phenotypeAsya L Borikova, Christopher F Dibble, Noah Sciaky, et al.
Human Molecular Genetics|July 23, 2005
CCM1 and CCM2 protein interactions in cell signaling: implications for cerebral cavernous malformations pathogenesisJon S Zawistowski, Lisa Stalheim, Mark T Uhlik, et al.
The Journal of Clinical Endocrinology and Metabolism|July 14, 1998
A unique constitutively activating mutation in third transmembrane helix of luteinizing hormone receptor causes sporadic male gonadotropin-independent precocious pubertyA C Latronico, A N Abell, I J Arnhold, et al.
Pageof 4