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N Aronin

Showing results (51-60 of 67) with videos related to

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Annals of Internal Medicine|July 1, 1984
Environmental iodine intake and thyroid dysfunction during chronic amiodarone therapyE Martino, M Safran, F Aghini-Lombardi, et al.
Neuron|November 1, 1995
CAG expansion affects the expression of mutant Huntingtin in the Huntington's disease brainN Aronin, K Chase, C Young, et al.
Journal of Neuropathology and Experimental Neurology|March 29, 2001
Early and progressive accumulation of reactive microglia in the Huntington disease brainE Sapp, K B Kegel, N Aronin, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 25, 2001
Caspase 3-cleaved N-terminal fragments of wild-type and mutant huntingtin are present in normal and Huntington's disease brains, associate with membranes, and undergo calpain-dependent proteolysisY J Kim, Y Yi, E Sapp, et al.
Journal of Cellular Biochemistry|June 1, 1992
Downregulation of histone H4 gene transcription during postnatal development in transgenic mice and at the onset of differentiation in transgenically derived calvarial osteoblast culturesS P Gerbaulet, A J van Wijnen, N Aronin, et al.
Annals of Neurology|October 24, 1997
Huntingtin localization in brains of normal and Huntington's disease patientsE Sapp, C Schwarz, K Chase, et al.
Molecular Pharmacology|June 1, 1990
Fluorescent and biotin probes for dopamine receptors: D1 and D2 receptor affinity and selectivityB K Madras, D R Canfield, C Pfaelzer, et al.
Neuroscience|June 11, 1999
Forskolin and dopamine D1 receptor activation increase huntingtin's association with endosomes in immortalized neuronal cells of striatal originM Kim, J Velier, K Chase, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 1, 1996
Expression of normal and mutant huntingtin in the developing brainP G Bhide, M Day, E Sapp, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 1, 1990
Coordinate occupancy of AP-1 sites in the vitamin D-responsive and CCAAT box elements by Fos-Jun in the osteocalcin gene: model for phenotype suppression of transcriptionT A Owen, R Bortell, S A Yocum, et al.
Pageof 7

Showing results (51-60 of 67) with videos related to

Sort By:
Pageof 7
Annals of Internal Medicine|July 1, 1984
Environmental iodine intake and thyroid dysfunction during chronic amiodarone therapyE Martino, M Safran, F Aghini-Lombardi, et al.
Neuron|November 1, 1995
CAG expansion affects the expression of mutant Huntingtin in the Huntington's disease brainN Aronin, K Chase, C Young, et al.
Journal of Neuropathology and Experimental Neurology|March 29, 2001
Early and progressive accumulation of reactive microglia in the Huntington disease brainE Sapp, K B Kegel, N Aronin, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 25, 2001
Caspase 3-cleaved N-terminal fragments of wild-type and mutant huntingtin are present in normal and Huntington's disease brains, associate with membranes, and undergo calpain-dependent proteolysisY J Kim, Y Yi, E Sapp, et al.
Journal of Cellular Biochemistry|June 1, 1992
Downregulation of histone H4 gene transcription during postnatal development in transgenic mice and at the onset of differentiation in transgenically derived calvarial osteoblast culturesS P Gerbaulet, A J van Wijnen, N Aronin, et al.
Annals of Neurology|October 24, 1997
Huntingtin localization in brains of normal and Huntington's disease patientsE Sapp, C Schwarz, K Chase, et al.
Molecular Pharmacology|June 1, 1990
Fluorescent and biotin probes for dopamine receptors: D1 and D2 receptor affinity and selectivityB K Madras, D R Canfield, C Pfaelzer, et al.
Neuroscience|June 11, 1999
Forskolin and dopamine D1 receptor activation increase huntingtin's association with endosomes in immortalized neuronal cells of striatal originM Kim, J Velier, K Chase, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 1, 1996
Expression of normal and mutant huntingtin in the developing brainP G Bhide, M Day, E Sapp, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 1, 1990
Coordinate occupancy of AP-1 sites in the vitamin D-responsive and CCAAT box elements by Fos-Jun in the osteocalcin gene: model for phenotype suppression of transcriptionT A Owen, R Bortell, S A Yocum, et al.
Pageof 7