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American Journal of Human Genetics|March 12, 2013
A CpG mutational hotspot in a ONECUT binding site accounts for the prevalent variant of hemophilia B LeydenAlister P W Funnell, Michael D Wilson, Benoit Ballester, et al.Journal of Immunology (Baltimore, Md. : 1950)|October 18, 2011
Impaired B cell development in the absence of Krüppel-like factor 3Thi Thanh Vu, Dominique Gatto, Vivian Turner, et al.BMC Molecular Biology|June 3, 2014
Differential regulation of the α-globin locus by Krüppel-like Factor 3 in erythroid and non-erythroid cellsAlister P W Funnell, Douglas Vernimmen, Wooi F Lim, et al.Molecular and Cellular Biology|April 9, 2008
Targeted disruption of the basic Krüppel-like factor gene (Klf3) reveals a role in adipogenesisNancy Sue, Briony H A Jack, Sally A Eaton, et al.Molecular and Cellular Biology|May 30, 2013
Generation of mice deficient in both KLF3/BKLF and KLF8 reveals a genetic interaction and a role for these factors in embryonic globin gene silencingAlister P W Funnell, Ka Sin Mak, Natalie A Twine, et al.The Journal of Biological Chemistry|February 10, 2015
Phosphorylation of Krüppel-like factor 3 (KLF3/BKLF) and C-terminal binding protein 2 (CtBP2) by homeodomain-interacting protein kinase 2 (HIPK2) modulates KLF3 DNA binding and activityVitri Dewi, Alister Kwok, Stella Lee, et al.Nature Chemistry|July 7, 2022
Structure-specific amyloid precipitation in biofluidsM Rodrigues, P Bhattacharjee, A Brinkmalm, et al.Molecular and Cellular Biology|June 20, 2012
The CACCC-binding protein KLF3/BKLF represses a subset of KLF1/EKLF target genes and is required for proper erythroid maturation in vivoAlister P W Funnell, Laura J Norton, Ka Sin Mak, et al.Pageof 8