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N E Reist

Showing results (1-10 of 11) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|August 15, 1992
Neurally evoked calcium transients in terminal Schwann cells at the neuromuscular junctionN E Reist, S J Smith
The Journal of Comparative Neurology|June 20, 2001
Mutations in the second C2 domain of synaptotagmin disrupt synaptic transmission at Drosophila neuromuscular junctionsJ M Mackler, N E Reist
The Journal of Cell Biology|December 1, 1987
Agrin-like molecules at synaptic sites in normal, denervated, and damaged skeletal musclesN E Reist, C Magill, U J McMahan
Genesis (New York, N.Y. : 2000)|October 23, 2001
Drosophila synaptotagmin I null mutants survive to early adulthoodC A Loewen, J M Mackler, N E Reist
Neuron|May 1, 1992
Agrin released by motor neurons induces the aggregation of acetylcholine receptors at neuromuscular junctionsN E Reist, M J Werle, U J McMahan
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 19, 1998
Morphologically docked synaptic vesicles are reduced in synaptotagmin mutants of DrosophilaN E Reist, J Buchanan, J Li, et al.
Nature|July 12, 2002
The C(2)B Ca(2+)-binding motif of synaptotagmin is required for synaptic transmission in vivoJ M Mackler, J A Drummond, C A Loewen, et al.
Nature|June 13, 1985
Acetylcholine receptor-aggregating factor is similar to molecules concentrated at neuromuscular junctionsJ R Fallon, R M Nitkin, N E Reist, et al.
The Journal of Experimental Biology|September 1, 1987
Identification of agrin in electric organ extracts and localization of agrin-like molecules in muscle and central nervous systemM A Smith, Y M Yao, N E Reist, et al.
Progress in Brain Research|January 1, 1987
AgrinC Magill, N E Reist, J R Fallon, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Proceedings of the National Academy of Sciences of the United States of America|August 15, 1992
Neurally evoked calcium transients in terminal Schwann cells at the neuromuscular junctionN E Reist, S J Smith
The Journal of Comparative Neurology|June 20, 2001
Mutations in the second C2 domain of synaptotagmin disrupt synaptic transmission at Drosophila neuromuscular junctionsJ M Mackler, N E Reist
The Journal of Cell Biology|December 1, 1987
Agrin-like molecules at synaptic sites in normal, denervated, and damaged skeletal musclesN E Reist, C Magill, U J McMahan
Genesis (New York, N.Y. : 2000)|October 23, 2001
Drosophila synaptotagmin I null mutants survive to early adulthoodC A Loewen, J M Mackler, N E Reist
Neuron|May 1, 1992
Agrin released by motor neurons induces the aggregation of acetylcholine receptors at neuromuscular junctionsN E Reist, M J Werle, U J McMahan
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 19, 1998
Morphologically docked synaptic vesicles are reduced in synaptotagmin mutants of DrosophilaN E Reist, J Buchanan, J Li, et al.
Nature|July 12, 2002
The C(2)B Ca(2+)-binding motif of synaptotagmin is required for synaptic transmission in vivoJ M Mackler, J A Drummond, C A Loewen, et al.
Nature|June 13, 1985
Acetylcholine receptor-aggregating factor is similar to molecules concentrated at neuromuscular junctionsJ R Fallon, R M Nitkin, N E Reist, et al.
The Journal of Experimental Biology|September 1, 1987
Identification of agrin in electric organ extracts and localization of agrin-like molecules in muscle and central nervous systemM A Smith, Y M Yao, N E Reist, et al.
Progress in Brain Research|January 1, 1987
AgrinC Magill, N E Reist, J R Fallon, et al.
Pageof 2