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N Gretz

Showing results (61-70 of 160) with videos related to

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American Journal of Physiology. Renal Physiology|February 22, 2001
A possible role for metalloproteinases in renal cyst developmentN Obermüller, N Morente, B Kränzlin, et al.
Medizinische Klinik (Munich, Germany : 1983)|April 15, 1993
[Autoregulation of kidney circulation, glomerular filtration rate and plasma renin activity in spontaneously hypertensive rats and normotensive Wistar rats]P Wende, M Strauch, T Unger, et al.
In Vitro Cellular & Developmental Biology. Animal|December 30, 1999
A new in vitro bioassay for cyst formation by renal cells from an autosomal dominant rat model of polycystic kidney diseaseR Pey, J Bach, G Schieren, et al.
Lancet (London, England)|July 15, 1989
Donor kidneys of infants and very young children are unacceptable for transplantationH Ruder, F Schaefer, N Gretz, et al.
Oncogene|August 2, 2011
Absent in Melanoma 2 (AIM2) is an important mediator of interferon-dependent and -independent HLA-DRA and HLA-DRB gene expression in colorectal cancersJ Lee, L Li, N Gretz, et al.
European Journal of Clinical Investigation|July 1, 1993
Chronic lead exposure in rats: effects on blood pressureR Nowack, A Wiecek, B Exner, et al.
Experimental Nephrology|May 1, 1994
Focal overexpression of collagen IV characterizes the initiation of epithelial changes in polycystic kidney diseaseK Schäfer, M Bader, N Gretz, et al.
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|September 1, 1989
Is gender a determinant for evolution of renal failure? A study in autosomal dominant polycystic kidney diseaseN Gretz, M Zeier, S Geberth, et al.
Pharmacological Research|May 16, 2015
Nephrotoxicity of ibandronate and zoledronate in Wistar rats with normal renal function and after unilateral nephrectomyR Bergner, B Siegrist, N Gretz, et al.
Fortschritte Der Neurologie-Psychiatrie|March 1, 1990
[Neuromyeloencephalopathy caused by high-dose muzolimine medication in patients with renal failure]B Pohlmann-Eden, P Berlit, A Holle, et al.
Pageof 16

Showing results (61-70 of 160) with videos related to

Sort By:
Pageof 16
American Journal of Physiology. Renal Physiology|February 22, 2001
A possible role for metalloproteinases in renal cyst developmentN Obermüller, N Morente, B Kränzlin, et al.
Medizinische Klinik (Munich, Germany : 1983)|April 15, 1993
[Autoregulation of kidney circulation, glomerular filtration rate and plasma renin activity in spontaneously hypertensive rats and normotensive Wistar rats]P Wende, M Strauch, T Unger, et al.
In Vitro Cellular & Developmental Biology. Animal|December 30, 1999
A new in vitro bioassay for cyst formation by renal cells from an autosomal dominant rat model of polycystic kidney diseaseR Pey, J Bach, G Schieren, et al.
Lancet (London, England)|July 15, 1989
Donor kidneys of infants and very young children are unacceptable for transplantationH Ruder, F Schaefer, N Gretz, et al.
Oncogene|August 2, 2011
Absent in Melanoma 2 (AIM2) is an important mediator of interferon-dependent and -independent HLA-DRA and HLA-DRB gene expression in colorectal cancersJ Lee, L Li, N Gretz, et al.
European Journal of Clinical Investigation|July 1, 1993
Chronic lead exposure in rats: effects on blood pressureR Nowack, A Wiecek, B Exner, et al.
Experimental Nephrology|May 1, 1994
Focal overexpression of collagen IV characterizes the initiation of epithelial changes in polycystic kidney diseaseK Schäfer, M Bader, N Gretz, et al.
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|September 1, 1989
Is gender a determinant for evolution of renal failure? A study in autosomal dominant polycystic kidney diseaseN Gretz, M Zeier, S Geberth, et al.
Pharmacological Research|May 16, 2015
Nephrotoxicity of ibandronate and zoledronate in Wistar rats with normal renal function and after unilateral nephrectomyR Bergner, B Siegrist, N Gretz, et al.
Fortschritte Der Neurologie-Psychiatrie|March 1, 1990
[Neuromyeloencephalopathy caused by high-dose muzolimine medication in patients with renal failure]B Pohlmann-Eden, P Berlit, A Holle, et al.
Pageof 16