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N J Wandersee

Showing results (1-10 of 10) with videos related to

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Current Opinion in Hematology|March 24, 1999
Thrombosis in heritable hemolytic disordersJ E Barker, N J Wandersee
Experimental Hematology|September 16, 2000
Amelioration of severe hereditary spherocytosis in nonablated adult mice by marrow transplantationJ E Barker, S Deveau, N J Wandersee
Blood Cells, Molecules & Diseases|April 25, 2000
Erythroid phosphatidyl serine exposure is not predictive of thrombotic risk in mice with hemolytic anemiaN J Wandersee, J F Tait, J E Barker
Molecular and Cellular Biology|January 1, 1996
Intronic and flanking sequences are required to silence enhancement of an embryonic beta-type globin geneN J Wandersee, R C Ferris, G D Ginder
Molecular and Cellular Biology|September 1, 1991
5'-flanking sequences mediate butyrate stimulation of embryonic globin gene expression in adult erythroid cellsJ G Glauber, N J Wandersee, J A Little, et al.
Blood|December 31, 1997
Thrombosis and secondary hemochromatosis play major roles in the pathogenesis of jaundiced and spherocytic mice, murine models for hereditary spherocytosisT M Kaysser, N J Wandersee, R T Bronson, et al.
Blood|June 5, 2001
Reduced incidence of thrombosis in mice with hereditary spherocytosis following neonatal treatment with normal hematopoietic cellsN J Wandersee, J C Lee, S A Deveau, et al.
The Hematology Journal : the Official Journal of the European Haematology Association|March 29, 2002
Murine recessive hereditary spherocytosis, sph/sph, is caused by a mutation in the erythroid alpha-spectrin geneN J Wandersee, C S Birkenmeier, E J Gifford, et al.
Blood|December 9, 1998
Hematopoietic cells from -spectrin-deficient mice are sufficient to induce thrombotic events in hematopoietically ablated recipientsN J Wandersee, J C Lee, T M Kaysser, et al.
Blood|January 12, 2001
Defective spectrin integrity and neonatal thrombosis in the first mouse model for severe hereditary elliptocytosisN J Wandersee, A N Roesch, N R Hamblen, et al.
Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:
Pageof 1
Current Opinion in Hematology|March 24, 1999
Thrombosis in heritable hemolytic disordersJ E Barker, N J Wandersee
Experimental Hematology|September 16, 2000
Amelioration of severe hereditary spherocytosis in nonablated adult mice by marrow transplantationJ E Barker, S Deveau, N J Wandersee
Blood Cells, Molecules & Diseases|April 25, 2000
Erythroid phosphatidyl serine exposure is not predictive of thrombotic risk in mice with hemolytic anemiaN J Wandersee, J F Tait, J E Barker
Molecular and Cellular Biology|January 1, 1996
Intronic and flanking sequences are required to silence enhancement of an embryonic beta-type globin geneN J Wandersee, R C Ferris, G D Ginder
Molecular and Cellular Biology|September 1, 1991
5'-flanking sequences mediate butyrate stimulation of embryonic globin gene expression in adult erythroid cellsJ G Glauber, N J Wandersee, J A Little, et al.
Blood|December 31, 1997
Thrombosis and secondary hemochromatosis play major roles in the pathogenesis of jaundiced and spherocytic mice, murine models for hereditary spherocytosisT M Kaysser, N J Wandersee, R T Bronson, et al.
Blood|June 5, 2001
Reduced incidence of thrombosis in mice with hereditary spherocytosis following neonatal treatment with normal hematopoietic cellsN J Wandersee, J C Lee, S A Deveau, et al.
The Hematology Journal : the Official Journal of the European Haematology Association|March 29, 2002
Murine recessive hereditary spherocytosis, sph/sph, is caused by a mutation in the erythroid alpha-spectrin geneN J Wandersee, C S Birkenmeier, E J Gifford, et al.
Blood|December 9, 1998
Hematopoietic cells from -spectrin-deficient mice are sufficient to induce thrombotic events in hematopoietically ablated recipientsN J Wandersee, J C Lee, T M Kaysser, et al.
Blood|January 12, 2001
Defective spectrin integrity and neonatal thrombosis in the first mouse model for severe hereditary elliptocytosisN J Wandersee, A N Roesch, N R Hamblen, et al.
Pageof 1